Endogenous formaldehyde as a potential factor of vulnerability of atherosclerosis: involvement of semicarbazide-sensitive amine oxidase-mediated methylamine turnover

The mouse is known to be highly resistant to atherosclerosis. However, some inbred mouse strains are vulnerable to atherosclerosis when they are fed a high-cholesterol, high-fat diet. Increased deamination of methylamine (MA) and the subsequent production of formaldehyde has been recently shown to b...

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Veröffentlicht in:	Atherosclerosis 1998-10, Vol.140 (2), p.357-363
Hauptverfasser:	Yu, P.H., Deng, Y.L.
Format:	Artikel
Sprache:	eng
Schlagworte:	Acetaldehyde Allyl Compounds - pharmacology Amine oxidase Amine Oxidase (Copper-Containing) - antagonists & inhibitors Amine Oxidase (Copper-Containing) - metabolism Animals Aorta - metabolism Arteriosclerosis - etiology Arteriosclerosis - metabolism Arteriosclerosis - prevention & control Atherosclerosis (general aspects, experimental research) Biological and medical sciences Blood and lymphatic vessels Butylamines - pharmacology Cardiology. Vascular system Chromatography, High Pressure Liquid Diet, Atherogenic Disease Susceptibility - metabolism Enzyme Inhibitors - pharmacology Formaldehyde Formaldehyde - metabolism Guanidines - pharmacology Kidney - metabolism Male Medical sciences Methylamine Methylamines - metabolism Methylglyoxal Mice Mice, Inbred BALB C Mice, Inbred C57BL Risk Factors SSAO
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