Atherosclerosis-prone hemodynamics differentially regulates endothelial and smooth muscle cell phenotypes and promotes pro-inflammatory priming

1 Department of Biomedical Engineering, 2 Robert M. Berne Cardiovascular Research Center, 3 Cardiovascular Division, Department of Medicine, and 4 Laboratory of Atherogenesis, University of Virginia, Charlottesville, Virginia Submitted 24 August 2007 ; accepted in final form 3 October 2007 Atheroscl...

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Veröffentlicht in:	American Journal of Physiology: Cell Physiology 2007-12, Vol.293 (6), p.C1824-C1833
Hauptverfasser:	Hastings, Nicole E, Simmers, Michael B, McDonald, Oliver G, Wamhoff, Brian R, Blackman, Brett R
Format:	Artikel
Sprache:	eng
Schlagworte:	Arteries - physiopathology Atherosclerosis - pathology Atherosclerosis - physiopathology Cell culture Cells, Cultured Chromatin Coculture Techniques Endothelial Cells - pathology Endothelium, Vascular - pathology Endothelium, Vascular - physiopathology Epigenesis, Genetic Gene Expression Genotype & phenotype Hemodynamics - physiology Humans Inflammation - physiopathology Inflammatory diseases Muscular system Myocytes, Smooth Muscle - pathology Phenotype Proteins - isolation & purification RNA - isolation & purification Shear stress
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