Arterial elasticity identified by pulse wave analysis and its relation to endothelial function in patients with coronary artery disease
Patients with coronary artery disease (CAD) have impaired endothelial function. Arterial elasticity is modulated by endothelial function. The association between arterial elasticity and endothelial function has not been reported in patients with CAD. The present study was designed to investigate whe...
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| Veröffentlicht in: | Journal of human hypertension 2007-02, Vol.21 (2), p.149-153 |
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| creator | Tao, J Liu, D-H Wang, L-C Wang, J-M Wang, Y Yang, Z Lou, Z-F Tang, A-L |
| description | Patients with coronary artery disease (CAD) have impaired endothelial function. Arterial elasticity is modulated by endothelial function. The association between arterial elasticity and endothelial function has not been reported in patients with CAD. The present study was designed to investigate whether endothelial dysfunction contributes to impaired arterial elasticity. Thirty patients with CAD and 30 control subjects were recruited. Large and small artery elasticity indices were non-invasively assessed using pulse wave analysis. Brachial artery endothelium-dependent and -independent function were assessed by vascular response to flow-mediated vasodilation (FMD) and sublingual nitroglyceride (NTG), respectively. C1 large artery elasticity index was not different in the CAD group compared with the control group. However, C2 small artery elasticity index was significantly reduced in the CAD group compared with the control group. Flow-mediated vasodilation (FMD) was also impaired in the CAD group compared with the control group. Flow-mediated vasodilation (FMD) in the brachial artery correlated with C2 small arterial elasticity index. But NTG-mediated brachial artery vasodilation was similar between the two groups. The present findings suggest that the patients with CAD have reduced C2 small arterial elasticity index and impaired FMD. Endothelial dysfunction is involved in diminished arterial elasticity, suggesting that C2 small arterial elasticity index is a novel surrogate measure for the clinical evaluation of endothelial function. |
| doi_str_mv | 10.1038/sj.jhh.1002112 |
| format | Article |
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Arterial elasticity is modulated by endothelial function. The association between arterial elasticity and endothelial function has not been reported in patients with CAD. The present study was designed to investigate whether endothelial dysfunction contributes to impaired arterial elasticity. Thirty patients with CAD and 30 control subjects were recruited. Large and small artery elasticity indices were non-invasively assessed using pulse wave analysis. Brachial artery endothelium-dependent and -independent function were assessed by vascular response to flow-mediated vasodilation (FMD) and sublingual nitroglyceride (NTG), respectively. C1 large artery elasticity index was not different in the CAD group compared with the control group. However, C2 small artery elasticity index was significantly reduced in the CAD group compared with the control group. Flow-mediated vasodilation (FMD) was also impaired in the CAD group compared with the control group. Flow-mediated vasodilation (FMD) in the brachial artery correlated with C2 small arterial elasticity index. But NTG-mediated brachial artery vasodilation was similar between the two groups. The present findings suggest that the patients with CAD have reduced C2 small arterial elasticity index and impaired FMD. Endothelial dysfunction is involved in diminished arterial elasticity, suggesting that C2 small arterial elasticity index is a novel surrogate measure for the clinical evaluation of endothelial function.</description><identifier>ISSN: 0950-9240</identifier><identifier>EISSN: 1476-5527</identifier><identifier>DOI: 10.1038/sj.jhh.1002112</identifier><identifier>PMID: 17082797</identifier><language>eng</language><publisher>London: Nature Publishing Group UK</publisher><subject>Aged ; Arterial hypertension. Arterial hypotension ; Arteries ; Arteries - physiopathology ; Biological and medical sciences ; Blood and lymphatic vessels ; Brachial Artery - physiopathology ; Cardiology. Vascular system ; Cardiovascular disease ; Clinical manifestations. Epidemiology. Investigative techniques. Etiology ; Coronary artery ; Coronary Disease - physiopathology ; Coronary heart disease ; Coronary vessels ; Elasticity ; Endothelium ; Endothelium, Vascular - physiopathology ; Epidemiology ; Female ; Health Administration ; Health aspects ; Heart ; Heart diseases ; Humans ; Linear Models ; Male ; Medical sciences ; Medicine ; Medicine & Public Health ; Middle Aged ; Nitric oxide ; original-article ; Patients ; Physiological aspects ; Public Health ; Pulsatile Flow - physiology ; Risk factors ; Vasodilation ; Vein & artery diseases</subject><ispartof>Journal of human hypertension, 2007-02, Vol.21 (2), p.149-153</ispartof><rights>Springer Nature Limited 2007</rights><rights>2007 INIST-CNRS</rights><rights>COPYRIGHT 2007 Nature Publishing Group</rights><rights>Copyright Nature Publishing Group Feb 2007</rights><rights>Nature Publishing Group 2007.</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c578t-9a683f0b48ca78bb7d1e8b605f3452c46c9be643c91973ce41da28f33e9fa8e93</citedby><cites>FETCH-LOGICAL-c578t-9a683f0b48ca78bb7d1e8b605f3452c46c9be643c91973ce41da28f33e9fa8e93</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://link.springer.com/content/pdf/10.1038/sj.jhh.1002112$$EPDF$$P50$$Gspringer$$H</linktopdf><linktohtml>$$Uhttps://link.springer.com/10.1038/sj.jhh.1002112$$EHTML$$P50$$Gspringer$$H</linktohtml><link.rule.ids>314,780,784,27924,27925,41488,42557,51319</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=18495561$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/17082797$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Tao, J</creatorcontrib><creatorcontrib>Liu, D-H</creatorcontrib><creatorcontrib>Wang, L-C</creatorcontrib><creatorcontrib>Wang, J-M</creatorcontrib><creatorcontrib>Wang, Y</creatorcontrib><creatorcontrib>Yang, Z</creatorcontrib><creatorcontrib>Lou, Z-F</creatorcontrib><creatorcontrib>Tang, A-L</creatorcontrib><title>Arterial elasticity identified by pulse wave analysis and its relation to endothelial function in patients with coronary artery disease</title><title>Journal of human hypertension</title><addtitle>J Hum Hypertens</addtitle><addtitle>J Hum Hypertens</addtitle><description>Patients with coronary artery disease (CAD) have impaired endothelial function. Arterial elasticity is modulated by endothelial function. The association between arterial elasticity and endothelial function has not been reported in patients with CAD. The present study was designed to investigate whether endothelial dysfunction contributes to impaired arterial elasticity. Thirty patients with CAD and 30 control subjects were recruited. Large and small artery elasticity indices were non-invasively assessed using pulse wave analysis. Brachial artery endothelium-dependent and -independent function were assessed by vascular response to flow-mediated vasodilation (FMD) and sublingual nitroglyceride (NTG), respectively. C1 large artery elasticity index was not different in the CAD group compared with the control group. However, C2 small artery elasticity index was significantly reduced in the CAD group compared with the control group. Flow-mediated vasodilation (FMD) was also impaired in the CAD group compared with the control group. Flow-mediated vasodilation (FMD) in the brachial artery correlated with C2 small arterial elasticity index. But NTG-mediated brachial artery vasodilation was similar between the two groups. The present findings suggest that the patients with CAD have reduced C2 small arterial elasticity index and impaired FMD. Endothelial dysfunction is involved in diminished arterial elasticity, suggesting that C2 small arterial elasticity index is a novel surrogate measure for the clinical evaluation of endothelial function.</description><subject>Aged</subject><subject>Arterial hypertension. Arterial hypotension</subject><subject>Arteries</subject><subject>Arteries - physiopathology</subject><subject>Biological and medical sciences</subject><subject>Blood and lymphatic vessels</subject><subject>Brachial Artery - physiopathology</subject><subject>Cardiology. Vascular system</subject><subject>Cardiovascular disease</subject><subject>Clinical manifestations. Epidemiology. Investigative techniques. Etiology</subject><subject>Coronary artery</subject><subject>Coronary Disease - physiopathology</subject><subject>Coronary heart disease</subject><subject>Coronary vessels</subject><subject>Elasticity</subject><subject>Endothelium</subject><subject>Endothelium, Vascular - physiopathology</subject><subject>Epidemiology</subject><subject>Female</subject><subject>Health Administration</subject><subject>Health aspects</subject><subject>Heart</subject><subject>Heart diseases</subject><subject>Humans</subject><subject>Linear Models</subject><subject>Male</subject><subject>Medical sciences</subject><subject>Medicine</subject><subject>Medicine & Public Health</subject><subject>Middle Aged</subject><subject>Nitric oxide</subject><subject>original-article</subject><subject>Patients</subject><subject>Physiological aspects</subject><subject>Public Health</subject><subject>Pulsatile Flow - physiology</subject><subject>Risk factors</subject><subject>Vasodilation</subject><subject>Vein & artery diseases</subject><issn>0950-9240</issn><issn>1476-5527</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2007</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><sourceid>ABUWG</sourceid><sourceid>AFKRA</sourceid><sourceid>AZQEC</sourceid><sourceid>BENPR</sourceid><sourceid>CCPQU</sourceid><sourceid>DWQXO</sourceid><sourceid>GNUQQ</sourceid><recordid>eNp1kk1v1DAQhiMEokvhyg1kUdHbbu04ie3jqiofUiUucI4cZ9J45bUXj0O1v4C_jcOutAW18sEjz_O-Y4-nKN4yumKUyyvcrDbjmGNaMlY-KxasEs2yrkvxvFhQVdOlKit6VrxC3FA6J-XL4owJKkuhxKL4vY4JotWOgNOYrLFpT2wPPtnBQk-6PdlNDoHc619AtNdujxZz0BObkMSsSjZ4kgIB34c0gpvNhsmbv-fWk10msh-Se5tGYkIMXsc90XPhPektgkZ4XbwYdK7z5rifFz8-3Xy__rK8_fb56_X6dmlqIdNS6UbygXaVNFrIrhM9A9k1tB54VZemaozqoKm4UUwJbqBivS7lwDmoQUtQ_Ly4PPjuYvg5AaZ2a9GAc9pDmLBtFKXZqs7gxX_gJkwxvx_bsqlo0zBFq0x9eJJiSjVC8epkdacdtNYPIUVt5rrtmknOuahFmanVI1RePWytCR4Gm8__EVw-EIygXRoxuGnuOz7qbGJAjDC0u2i3-Q9aRtt5ilrctHmK2uMUZcH746umbgv9CT-OTQY-HgGNRrsham8snjhZqbpuWOauDhzmlL-DeGrPk6XfHRRepynCg5se8n8AiwDpWg</recordid><startdate>20070201</startdate><enddate>20070201</enddate><creator>Tao, J</creator><creator>Liu, D-H</creator><creator>Wang, L-C</creator><creator>Wang, J-M</creator><creator>Wang, Y</creator><creator>Yang, Z</creator><creator>Lou, Z-F</creator><creator>Tang, A-L</creator><general>Nature Publishing Group UK</general><general>Nature Publishing</general><general>Nature Publishing Group</general><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>3V.</scope><scope>7T5</scope><scope>7X7</scope><scope>7XB</scope><scope>88E</scope><scope>8AO</scope><scope>8FE</scope><scope>8FH</scope><scope>8FI</scope><scope>8FJ</scope><scope>8FK</scope><scope>ABUWG</scope><scope>AFKRA</scope><scope>AZQEC</scope><scope>BBNVY</scope><scope>BENPR</scope><scope>BHPHI</scope><scope>CCPQU</scope><scope>DWQXO</scope><scope>FYUFA</scope><scope>GHDGH</scope><scope>GNUQQ</scope><scope>H94</scope><scope>HCIFZ</scope><scope>K9.</scope><scope>LK8</scope><scope>M0S</scope><scope>M1P</scope><scope>M7P</scope><scope>PQEST</scope><scope>PQQKQ</scope><scope>PQUKI</scope><scope>PRINS</scope><scope>7X8</scope></search><sort><creationdate>20070201</creationdate><title>Arterial elasticity identified by pulse wave analysis and its relation to endothelial function in patients with coronary artery disease</title><author>Tao, J ; Liu, D-H ; Wang, L-C ; Wang, J-M ; Wang, Y ; Yang, Z ; Lou, Z-F ; Tang, A-L</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c578t-9a683f0b48ca78bb7d1e8b605f3452c46c9be643c91973ce41da28f33e9fa8e93</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2007</creationdate><topic>Aged</topic><topic>Arterial hypertension. Arterial hypotension</topic><topic>Arteries</topic><topic>Arteries - physiopathology</topic><topic>Biological and medical sciences</topic><topic>Blood and lymphatic vessels</topic><topic>Brachial Artery - physiopathology</topic><topic>Cardiology. Vascular system</topic><topic>Cardiovascular disease</topic><topic>Clinical manifestations. Epidemiology. Investigative techniques. Etiology</topic><topic>Coronary artery</topic><topic>Coronary Disease - physiopathology</topic><topic>Coronary heart disease</topic><topic>Coronary vessels</topic><topic>Elasticity</topic><topic>Endothelium</topic><topic>Endothelium, Vascular - physiopathology</topic><topic>Epidemiology</topic><topic>Female</topic><topic>Health Administration</topic><topic>Health aspects</topic><topic>Heart</topic><topic>Heart diseases</topic><topic>Humans</topic><topic>Linear Models</topic><topic>Male</topic><topic>Medical sciences</topic><topic>Medicine</topic><topic>Medicine & Public Health</topic><topic>Middle Aged</topic><topic>Nitric oxide</topic><topic>original-article</topic><topic>Patients</topic><topic>Physiological aspects</topic><topic>Public Health</topic><topic>Pulsatile Flow - physiology</topic><topic>Risk factors</topic><topic>Vasodilation</topic><topic>Vein & artery diseases</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Tao, J</creatorcontrib><creatorcontrib>Liu, D-H</creatorcontrib><creatorcontrib>Wang, L-C</creatorcontrib><creatorcontrib>Wang, J-M</creatorcontrib><creatorcontrib>Wang, Y</creatorcontrib><creatorcontrib>Yang, Z</creatorcontrib><creatorcontrib>Lou, Z-F</creatorcontrib><creatorcontrib>Tang, A-L</creatorcontrib><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>ProQuest Central (Corporate)</collection><collection>Immunology Abstracts</collection><collection>Health & Medical Collection</collection><collection>ProQuest Central (purchase pre-March 2016)</collection><collection>Medical Database (Alumni Edition)</collection><collection>ProQuest Pharma Collection</collection><collection>ProQuest SciTech Collection</collection><collection>ProQuest Natural Science Collection</collection><collection>Hospital Premium Collection</collection><collection>Hospital Premium Collection (Alumni Edition)</collection><collection>ProQuest Central (Alumni) (purchase pre-March 2016)</collection><collection>ProQuest Central (Alumni Edition)</collection><collection>ProQuest Central UK/Ireland</collection><collection>ProQuest Central Essentials</collection><collection>Biological Science Collection</collection><collection>ProQuest Central</collection><collection>Natural Science Collection</collection><collection>ProQuest One Community College</collection><collection>ProQuest Central Korea</collection><collection>Health Research Premium Collection</collection><collection>Health Research Premium Collection (Alumni)</collection><collection>ProQuest Central Student</collection><collection>AIDS and Cancer Research Abstracts</collection><collection>SciTech Premium Collection</collection><collection>ProQuest Health & Medical Complete (Alumni)</collection><collection>ProQuest Biological Science Collection</collection><collection>Health & Medical Collection (Alumni Edition)</collection><collection>Medical Database</collection><collection>Biological Science Database</collection><collection>ProQuest One Academic Eastern Edition (DO NOT USE)</collection><collection>ProQuest One Academic</collection><collection>ProQuest One Academic UKI Edition</collection><collection>ProQuest Central China</collection><collection>MEDLINE - Academic</collection><jtitle>Journal of human hypertension</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Tao, J</au><au>Liu, D-H</au><au>Wang, L-C</au><au>Wang, J-M</au><au>Wang, Y</au><au>Yang, Z</au><au>Lou, Z-F</au><au>Tang, A-L</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Arterial elasticity identified by pulse wave analysis and its relation to endothelial function in patients with coronary artery disease</atitle><jtitle>Journal of human hypertension</jtitle><stitle>J Hum Hypertens</stitle><addtitle>J Hum Hypertens</addtitle><date>2007-02-01</date><risdate>2007</risdate><volume>21</volume><issue>2</issue><spage>149</spage><epage>153</epage><pages>149-153</pages><issn>0950-9240</issn><eissn>1476-5527</eissn><abstract>Patients with coronary artery disease (CAD) have impaired endothelial function. Arterial elasticity is modulated by endothelial function. The association between arterial elasticity and endothelial function has not been reported in patients with CAD. The present study was designed to investigate whether endothelial dysfunction contributes to impaired arterial elasticity. Thirty patients with CAD and 30 control subjects were recruited. Large and small artery elasticity indices were non-invasively assessed using pulse wave analysis. Brachial artery endothelium-dependent and -independent function were assessed by vascular response to flow-mediated vasodilation (FMD) and sublingual nitroglyceride (NTG), respectively. C1 large artery elasticity index was not different in the CAD group compared with the control group. However, C2 small artery elasticity index was significantly reduced in the CAD group compared with the control group. Flow-mediated vasodilation (FMD) was also impaired in the CAD group compared with the control group. Flow-mediated vasodilation (FMD) in the brachial artery correlated with C2 small arterial elasticity index. But NTG-mediated brachial artery vasodilation was similar between the two groups. The present findings suggest that the patients with CAD have reduced C2 small arterial elasticity index and impaired FMD. Endothelial dysfunction is involved in diminished arterial elasticity, suggesting that C2 small arterial elasticity index is a novel surrogate measure for the clinical evaluation of endothelial function.</abstract><cop>London</cop><pub>Nature Publishing Group UK</pub><pmid>17082797</pmid><doi>10.1038/sj.jhh.1002112</doi><tpages>5</tpages><oa>free_for_read</oa></addata></record> |
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| subjects | Aged Arterial hypertension. Arterial hypotension Arteries Arteries - physiopathology Biological and medical sciences Blood and lymphatic vessels Brachial Artery - physiopathology Cardiology. Vascular system Cardiovascular disease Clinical manifestations. Epidemiology. Investigative techniques. Etiology Coronary artery Coronary Disease - physiopathology Coronary heart disease Coronary vessels Elasticity Endothelium Endothelium, Vascular - physiopathology Epidemiology Female Health Administration Health aspects Heart Heart diseases Humans Linear Models Male Medical sciences Medicine Medicine & Public Health Middle Aged Nitric oxide original-article Patients Physiological aspects Public Health Pulsatile Flow - physiology Risk factors Vasodilation Vein & artery diseases |
| title | Arterial elasticity identified by pulse wave analysis and its relation to endothelial function in patients with coronary artery disease |
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