Crosstalk between EGFR and TrkB enhances ovarian cancer cell migration and proliferation

Ovarian cancer remains the leading cause of fatality among all gynecologic cancers, although promising therapies are in the making. It has been speculated that metastasis is critical for ovarian cancer, and yet the molecular mechanisms of metastasis in ovarian cancer are poorly understood. Growth fa...

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Veröffentlicht in:	International journal of oncology 2006-10, Vol.29 (4), p.1003-1011
Hauptverfasser:	LIHUA QIU, CHANGLIN ZHOU, YUN SUN, WEN DI, SCHEFFLER, Erica, HEALEY, Sarah, KOUTTAB, Nicola, WENMING CHU, YINSHENG WAN
Format:	Artikel
Sprache:	eng
Schlagworte:	Biological and medical sciences Brain-Derived Neurotrophic Factor - antagonists & inhibitors Brain-Derived Neurotrophic Factor - pharmacology Cell Movement - drug effects Cell Proliferation - drug effects Enzyme Inhibitors - pharmacology Epidermal Growth Factor - antagonists & inhibitors Epidermal Growth Factor - pharmacology Female Female genital diseases Gynecology. Andrology. Obstetrics Humans Medical sciences Ovarian Neoplasms - metabolism Ovarian Neoplasms - pathology Phosphatidylinositol 3-Kinases - antagonists & inhibitors Proto-Oncogene Proteins c-akt - agonists Proto-Oncogene Proteins c-akt - antagonists & inhibitors Receptor, Epidermal Growth Factor - agonists Receptor, Epidermal Growth Factor - genetics Receptor, Epidermal Growth Factor - metabolism Receptor, trkB - agonists Receptor, trkB - genetics Receptor, trkB - metabolism Transcriptional Activation Tumor Cells, Cultured Tumors
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