Parenteral iron treatment induces MCP-1 accumulation in plasma, normal kidneys, and in experimental nephropathy

Parenteral iron treatment induces MCP-1 accumulation in plasma, normal kidneys, and in experimental nephropathy. Monocyte chemoattractant protein-1 (MCP-1) promotes renal inflammation, thereby contributing to acute and chronic nephropathies. Its production is stimulated by oxidative stress. Thus, th...

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Veröffentlicht in:	Kidney international 2005-10, Vol.68 (4), p.1533-1542
1. Verfasser:	Zager, Richard A.
Format:	Artikel
Sprache:	eng
Schlagworte:	Acute Disease Animals Biological and medical sciences Cells, Cultured Chemokine CCL2 - blood Chemokine CCL2 - genetics Ferric Compounds - pharmacology Gene Expression - drug effects Glucaric Acid Gluconates - pharmacology Humans Injections, Intravenous iron dextran iron gluconate iron sucrose Iron-Dextran Complex - pharmacology Kidney Cortex - cytology Kidney Cortex - physiology Kidney Diseases - blood Kidney Diseases - physiopathology Liver - physiology Male Medical sciences Mice Mice, Inbred Strains Nephrology. Urinary tract diseases obstructive nephropathy oxidant stress Oxidative Stress - drug effects Peritoneum - physiology RNA, Messenger - analysis
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