GH-releasing peptides improve cardiac dysfunction and cachexia and suppress stress-related hormones and cardiomyocyte apoptosis in rats with heart failure

1 Department of Physiology and 3 National Laboratory of Medical Molecular Biology, School of Basic Medicine, Peking Union Medical College, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences; 2 Division of Cardiology, Department of Medicine, Peking Union Hospital, Beijing, China...

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Veröffentlicht in:	American journal of physiology. Heart and circulatory physiology 2005-10, Vol.289 (4), p.H1643-H1651
Hauptverfasser:	Xu, Xiang-Bin, Pang, Jin-Jiang, Cao, Ji-Min, Ni, Chao, Xu, Rong-Kun, Peng, Xiao-Zhong, Yu, Xiao-Xia, Guo, Shu, Chen, Meng-Chin, Chen, Chen
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Apoptosis - drug effects Blood Pressure - drug effects Cachexia - drug therapy Cachexia - pathology Catecholamines - blood Creatine Kinase - metabolism Heart Failure - drug therapy Heart Failure - pathology Hypophysectomy Myocardial Ischemia - drug therapy Myocardial Ischemia - pathology Myocytes, Cardiac - drug effects Myocytes, Cardiac - metabolism Myocytes, Cardiac - pathology Oligopeptides - pharmacology Rats Rats, Sprague-Dawley Receptors, G-Protein-Coupled - genetics Receptors, Ghrelin RNA, Messenger - analysis Ventricular Dysfunction, Left - drug therapy Ventricular Dysfunction, Left - pathology Ventricular Remodeling - drug effects
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