Reversal of cardiomyopathy in patients with repetitive monomorphic ventricular ectopy originating from the right ventricular outflow tract
Tachycardia-induced cardiomyopathy caused by ventricular tachycardia is a well-defined clinical entity. Less well appreciated is whether simple ventricular ectopy can result in cardiomyopathy. We sought to examine a potential causal relationship between repetitive monomorphic ventricular ectopy orig...
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| Veröffentlicht in: | Circulation (New York, N.Y.) N.Y.), 2005-08, Vol.112 (8), p.1092-1097 |
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| creator | YARLAGADDA, Ravi K IWAI, Sei STEIN, Kenneth M MARKOWITZ, Steven M SHAH, Bindi K CHEUNG, Jim W TAN, Vivian LERMAN, Bruce B MITTAL, Suneet |
| description | Tachycardia-induced cardiomyopathy caused by ventricular tachycardia is a well-defined clinical entity. Less well appreciated is whether simple ventricular ectopy can result in cardiomyopathy. We sought to examine a potential causal relationship between repetitive monomorphic ventricular ectopy originating from the right ventricular outflow tract and cardiomyopathy and the role of ablation in reversing this process.
The study consisted of 27 patients (11 men; age, 47+/-15 years) with repetitive monomorphic ventricular ectopy, including 8 patients (30%) with depressed ventricular function (ejection fraction < or =45%). All patients underwent assessment of cardiac structure and function. The burden of ectopy was quantified through 24-hour Holter monitoring. Patients then underwent ablation guided by 3D mapping. After ablation, patients underwent repeated Holter monitoring and reassessment of cardiac function. Patients with depressed ventricular function were more likely to be older than patients with normal function (58+/-14 versus 42+/-18 years; P=0.013). However, the burden of ventricular ectopy was similar in patients with (17,859+/-13,488 ectopic beats per 24 hours) and without (17,541+/-11,479 ectopic beats per 24 hours; P=0.800) preserved ventricular function. Successful ablation was performed in 23 patients (85%), including 7 of 8 patients with depressed ventricular function. In this latter group, ventricular function improved in all patients (from 39+/-6% to 62+/-6%; P=0.017).
Repetitive monomorphic ventricular ectopy (in the absence of sustained ventricular tachycardia) originating from the right ventricular outflow tract is an underappreciated cause of unexplained cardiomyopathy. Successful ablation of the focal source of ventricular ectopy results in normalization of left ventricular function. Patients with ectopy-induced cardiomyopathy are significantly older than patients with preserved ventricular function, which suggests either that older patients are more susceptible to the development of a cardiomyopathy or that the cardiomyopathy has had a longer period of time in which to evolve. |
| doi_str_mv | 10.1161/CIRCULATIONAHA.105.546432 |
| format | Article |
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The study consisted of 27 patients (11 men; age, 47+/-15 years) with repetitive monomorphic ventricular ectopy, including 8 patients (30%) with depressed ventricular function (ejection fraction < or =45%). All patients underwent assessment of cardiac structure and function. The burden of ectopy was quantified through 24-hour Holter monitoring. Patients then underwent ablation guided by 3D mapping. After ablation, patients underwent repeated Holter monitoring and reassessment of cardiac function. Patients with depressed ventricular function were more likely to be older than patients with normal function (58+/-14 versus 42+/-18 years; P=0.013). However, the burden of ventricular ectopy was similar in patients with (17,859+/-13,488 ectopic beats per 24 hours) and without (17,541+/-11,479 ectopic beats per 24 hours; P=0.800) preserved ventricular function. Successful ablation was performed in 23 patients (85%), including 7 of 8 patients with depressed ventricular function. In this latter group, ventricular function improved in all patients (from 39+/-6% to 62+/-6%; P=0.017).
Repetitive monomorphic ventricular ectopy (in the absence of sustained ventricular tachycardia) originating from the right ventricular outflow tract is an underappreciated cause of unexplained cardiomyopathy. Successful ablation of the focal source of ventricular ectopy results in normalization of left ventricular function. Patients with ectopy-induced cardiomyopathy are significantly older than patients with preserved ventricular function, which suggests either that older patients are more susceptible to the development of a cardiomyopathy or that the cardiomyopathy has had a longer period of time in which to evolve.</description><identifier>ISSN: 0009-7322</identifier><identifier>EISSN: 1524-4539</identifier><identifier>DOI: 10.1161/CIRCULATIONAHA.105.546432</identifier><identifier>PMID: 16103234</identifier><identifier>CODEN: CIRCAZ</identifier><language>eng</language><publisher>Hagerstown, MD: Lippincott Williams & Wilkins</publisher><subject>Adult ; Biological and medical sciences ; Blood and lymphatic vessels ; Cardiology. Vascular system ; Cardiomyopathies - diagnosis ; Cardiomyopathies - etiology ; Cardiomyopathies - therapy ; Cardiovascular system ; Catheter Ablation ; Diseases of the peripheral vessels. Diseases of the vena cava. Miscellaneous ; Electrocardiography, Ambulatory ; Female ; General and cellular metabolism. Vitamins ; Heart Conduction System ; Humans ; Male ; Medical sciences ; Middle Aged ; Pharmacology. Drug treatments ; Tachycardia, Ventricular - complications ; Tachycardia, Ventricular - diagnosis ; Tachycardia, Ventricular - therapy ; Vasodilator agents. Cerebral vasodilators ; Ventricular Dysfunction, Right - diagnosis ; Ventricular Dysfunction, Right - etiology ; Ventricular Dysfunction, Right - therapy ; Ventricular Premature Complexes - complications ; Ventricular Premature Complexes - diagnosis ; Ventricular Premature Complexes - therapy</subject><ispartof>Circulation (New York, N.Y.), 2005-08, Vol.112 (8), p.1092-1097</ispartof><rights>2005 INIST-CNRS</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c438t-f95ebd49d82c5dddc8399a438f03057c1fe7a6c23d9a471c5760513ace77547f3</citedby><cites>FETCH-LOGICAL-c438t-f95ebd49d82c5dddc8399a438f03057c1fe7a6c23d9a471c5760513ace77547f3</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>314,780,784,3687,27924,27925</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=17096815$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/16103234$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>YARLAGADDA, Ravi K</creatorcontrib><creatorcontrib>IWAI, Sei</creatorcontrib><creatorcontrib>STEIN, Kenneth M</creatorcontrib><creatorcontrib>MARKOWITZ, Steven M</creatorcontrib><creatorcontrib>SHAH, Bindi K</creatorcontrib><creatorcontrib>CHEUNG, Jim W</creatorcontrib><creatorcontrib>TAN, Vivian</creatorcontrib><creatorcontrib>LERMAN, Bruce B</creatorcontrib><creatorcontrib>MITTAL, Suneet</creatorcontrib><title>Reversal of cardiomyopathy in patients with repetitive monomorphic ventricular ectopy originating from the right ventricular outflow tract</title><title>Circulation (New York, N.Y.)</title><addtitle>Circulation</addtitle><description>Tachycardia-induced cardiomyopathy caused by ventricular tachycardia is a well-defined clinical entity. Less well appreciated is whether simple ventricular ectopy can result in cardiomyopathy. We sought to examine a potential causal relationship between repetitive monomorphic ventricular ectopy originating from the right ventricular outflow tract and cardiomyopathy and the role of ablation in reversing this process.
The study consisted of 27 patients (11 men; age, 47+/-15 years) with repetitive monomorphic ventricular ectopy, including 8 patients (30%) with depressed ventricular function (ejection fraction < or =45%). All patients underwent assessment of cardiac structure and function. The burden of ectopy was quantified through 24-hour Holter monitoring. Patients then underwent ablation guided by 3D mapping. After ablation, patients underwent repeated Holter monitoring and reassessment of cardiac function. Patients with depressed ventricular function were more likely to be older than patients with normal function (58+/-14 versus 42+/-18 years; P=0.013). However, the burden of ventricular ectopy was similar in patients with (17,859+/-13,488 ectopic beats per 24 hours) and without (17,541+/-11,479 ectopic beats per 24 hours; P=0.800) preserved ventricular function. Successful ablation was performed in 23 patients (85%), including 7 of 8 patients with depressed ventricular function. In this latter group, ventricular function improved in all patients (from 39+/-6% to 62+/-6%; P=0.017).
Repetitive monomorphic ventricular ectopy (in the absence of sustained ventricular tachycardia) originating from the right ventricular outflow tract is an underappreciated cause of unexplained cardiomyopathy. Successful ablation of the focal source of ventricular ectopy results in normalization of left ventricular function. Patients with ectopy-induced cardiomyopathy are significantly older than patients with preserved ventricular function, which suggests either that older patients are more susceptible to the development of a cardiomyopathy or that the cardiomyopathy has had a longer period of time in which to evolve.</description><subject>Adult</subject><subject>Biological and medical sciences</subject><subject>Blood and lymphatic vessels</subject><subject>Cardiology. Vascular system</subject><subject>Cardiomyopathies - diagnosis</subject><subject>Cardiomyopathies - etiology</subject><subject>Cardiomyopathies - therapy</subject><subject>Cardiovascular system</subject><subject>Catheter Ablation</subject><subject>Diseases of the peripheral vessels. Diseases of the vena cava. Miscellaneous</subject><subject>Electrocardiography, Ambulatory</subject><subject>Female</subject><subject>General and cellular metabolism. Vitamins</subject><subject>Heart Conduction System</subject><subject>Humans</subject><subject>Male</subject><subject>Medical sciences</subject><subject>Middle Aged</subject><subject>Pharmacology. Drug treatments</subject><subject>Tachycardia, Ventricular - complications</subject><subject>Tachycardia, Ventricular - diagnosis</subject><subject>Tachycardia, Ventricular - therapy</subject><subject>Vasodilator agents. Cerebral vasodilators</subject><subject>Ventricular Dysfunction, Right - diagnosis</subject><subject>Ventricular Dysfunction, Right - etiology</subject><subject>Ventricular Dysfunction, Right - therapy</subject><subject>Ventricular Premature Complexes - complications</subject><subject>Ventricular Premature Complexes - diagnosis</subject><subject>Ventricular Premature Complexes - therapy</subject><issn>0009-7322</issn><issn>1524-4539</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2005</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNpVkVFv2yAUhdG0ac3a_oWKPaxvzsCAsR-jqG0iRatUtc8WwZeYyjYe4FT5C_3VpUqkqE_A4TvnSvcg9JuSOaUF_btcPy1fNovn9eO_xWoxp0TMBS84y7-hGRU5z7hg1Xc0I4RUmWR5foF-hfCangWT4ie6SCGE5YzP0PsT7MEH1WFnsFa-sa4_uFHF9oDtgNPFwhADfrOxxR5GiDbaPeDeDa53fmytxvtEeKunTnkMOrrxgJ23Ozsk87DDxrsexxZw0tr4hXZTNJ17w9ErHa_QD6O6ANen8xK93N89L1fZ5vFhvVxsMs1ZGTNTCdg2vGrKXIumaXTJqkqlL0MYEVJTA1IVOmdNUiXVQhZEUKY0SCm4NOwS3R5zR-_-TxBi3dugoevUAG4KdVEKwmUhE1gdQe1dCB5MPXrbK3-oKak_i6i_FpFkUR-LSN6b05Bp20Nzdp42n4A_J0AFrTrj1aBtOHOSVEVJBfsArdyXlQ</recordid><startdate>20050823</startdate><enddate>20050823</enddate><creator>YARLAGADDA, Ravi K</creator><creator>IWAI, Sei</creator><creator>STEIN, Kenneth M</creator><creator>MARKOWITZ, Steven M</creator><creator>SHAH, Bindi K</creator><creator>CHEUNG, Jim W</creator><creator>TAN, Vivian</creator><creator>LERMAN, Bruce B</creator><creator>MITTAL, Suneet</creator><general>Lippincott Williams & Wilkins</general><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>20050823</creationdate><title>Reversal of cardiomyopathy in patients with repetitive monomorphic ventricular ectopy originating from the right ventricular outflow tract</title><author>YARLAGADDA, Ravi K ; IWAI, Sei ; STEIN, Kenneth M ; MARKOWITZ, Steven M ; SHAH, Bindi K ; CHEUNG, Jim W ; TAN, Vivian ; LERMAN, Bruce B ; MITTAL, Suneet</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c438t-f95ebd49d82c5dddc8399a438f03057c1fe7a6c23d9a471c5760513ace77547f3</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2005</creationdate><topic>Adult</topic><topic>Biological and medical sciences</topic><topic>Blood and lymphatic vessels</topic><topic>Cardiology. Vascular system</topic><topic>Cardiomyopathies - diagnosis</topic><topic>Cardiomyopathies - etiology</topic><topic>Cardiomyopathies - therapy</topic><topic>Cardiovascular system</topic><topic>Catheter Ablation</topic><topic>Diseases of the peripheral vessels. Diseases of the vena cava. Miscellaneous</topic><topic>Electrocardiography, Ambulatory</topic><topic>Female</topic><topic>General and cellular metabolism. Vitamins</topic><topic>Heart Conduction System</topic><topic>Humans</topic><topic>Male</topic><topic>Medical sciences</topic><topic>Middle Aged</topic><topic>Pharmacology. Drug treatments</topic><topic>Tachycardia, Ventricular - complications</topic><topic>Tachycardia, Ventricular - diagnosis</topic><topic>Tachycardia, Ventricular - therapy</topic><topic>Vasodilator agents. Cerebral vasodilators</topic><topic>Ventricular Dysfunction, Right - diagnosis</topic><topic>Ventricular Dysfunction, Right - etiology</topic><topic>Ventricular Dysfunction, Right - therapy</topic><topic>Ventricular Premature Complexes - complications</topic><topic>Ventricular Premature Complexes - diagnosis</topic><topic>Ventricular Premature Complexes - therapy</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>YARLAGADDA, Ravi K</creatorcontrib><creatorcontrib>IWAI, Sei</creatorcontrib><creatorcontrib>STEIN, Kenneth M</creatorcontrib><creatorcontrib>MARKOWITZ, Steven M</creatorcontrib><creatorcontrib>SHAH, Bindi K</creatorcontrib><creatorcontrib>CHEUNG, Jim W</creatorcontrib><creatorcontrib>TAN, Vivian</creatorcontrib><creatorcontrib>LERMAN, Bruce B</creatorcontrib><creatorcontrib>MITTAL, Suneet</creatorcontrib><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Circulation (New York, N.Y.)</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>YARLAGADDA, Ravi K</au><au>IWAI, Sei</au><au>STEIN, Kenneth M</au><au>MARKOWITZ, Steven M</au><au>SHAH, Bindi K</au><au>CHEUNG, Jim W</au><au>TAN, Vivian</au><au>LERMAN, Bruce B</au><au>MITTAL, Suneet</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Reversal of cardiomyopathy in patients with repetitive monomorphic ventricular ectopy originating from the right ventricular outflow tract</atitle><jtitle>Circulation (New York, N.Y.)</jtitle><addtitle>Circulation</addtitle><date>2005-08-23</date><risdate>2005</risdate><volume>112</volume><issue>8</issue><spage>1092</spage><epage>1097</epage><pages>1092-1097</pages><issn>0009-7322</issn><eissn>1524-4539</eissn><coden>CIRCAZ</coden><abstract>Tachycardia-induced cardiomyopathy caused by ventricular tachycardia is a well-defined clinical entity. Less well appreciated is whether simple ventricular ectopy can result in cardiomyopathy. We sought to examine a potential causal relationship between repetitive monomorphic ventricular ectopy originating from the right ventricular outflow tract and cardiomyopathy and the role of ablation in reversing this process.
The study consisted of 27 patients (11 men; age, 47+/-15 years) with repetitive monomorphic ventricular ectopy, including 8 patients (30%) with depressed ventricular function (ejection fraction < or =45%). All patients underwent assessment of cardiac structure and function. The burden of ectopy was quantified through 24-hour Holter monitoring. Patients then underwent ablation guided by 3D mapping. After ablation, patients underwent repeated Holter monitoring and reassessment of cardiac function. Patients with depressed ventricular function were more likely to be older than patients with normal function (58+/-14 versus 42+/-18 years; P=0.013). However, the burden of ventricular ectopy was similar in patients with (17,859+/-13,488 ectopic beats per 24 hours) and without (17,541+/-11,479 ectopic beats per 24 hours; P=0.800) preserved ventricular function. Successful ablation was performed in 23 patients (85%), including 7 of 8 patients with depressed ventricular function. In this latter group, ventricular function improved in all patients (from 39+/-6% to 62+/-6%; P=0.017).
Repetitive monomorphic ventricular ectopy (in the absence of sustained ventricular tachycardia) originating from the right ventricular outflow tract is an underappreciated cause of unexplained cardiomyopathy. Successful ablation of the focal source of ventricular ectopy results in normalization of left ventricular function. Patients with ectopy-induced cardiomyopathy are significantly older than patients with preserved ventricular function, which suggests either that older patients are more susceptible to the development of a cardiomyopathy or that the cardiomyopathy has had a longer period of time in which to evolve.</abstract><cop>Hagerstown, MD</cop><pub>Lippincott Williams & Wilkins</pub><pmid>16103234</pmid><doi>10.1161/CIRCULATIONAHA.105.546432</doi><tpages>6</tpages><oa>free_for_read</oa></addata></record> |
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| subjects | Adult Biological and medical sciences Blood and lymphatic vessels Cardiology. Vascular system Cardiomyopathies - diagnosis Cardiomyopathies - etiology Cardiomyopathies - therapy Cardiovascular system Catheter Ablation Diseases of the peripheral vessels. Diseases of the vena cava. Miscellaneous Electrocardiography, Ambulatory Female General and cellular metabolism. Vitamins Heart Conduction System Humans Male Medical sciences Middle Aged Pharmacology. Drug treatments Tachycardia, Ventricular - complications Tachycardia, Ventricular - diagnosis Tachycardia, Ventricular - therapy Vasodilator agents. Cerebral vasodilators Ventricular Dysfunction, Right - diagnosis Ventricular Dysfunction, Right - etiology Ventricular Dysfunction, Right - therapy Ventricular Premature Complexes - complications Ventricular Premature Complexes - diagnosis Ventricular Premature Complexes - therapy |
| title | Reversal of cardiomyopathy in patients with repetitive monomorphic ventricular ectopy originating from the right ventricular outflow tract |
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