Hepatitis C virus escape from the interferon regulatory factor 3 pathway by a passive and active evasion strategy

Hepatitis C virus (HCV) has been known to replicate with extremely varying efficiencies in different host cells, even within different populations of a single human hepatoma cell line, termed Huh‐7. Several reports have implicated the retinoic‐acid inducible gene I (RIG‐I)/ interferon regulatory fac...

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Veröffentlicht in:	Hepatology (Baltimore, Md.) Md.), 2007-11, Vol.46 (5), p.1365-1374
Hauptverfasser:	Binder, Marco, Kochs, Georg, Bartenschlager, Ralf, Lohmann, Volker
Format:	Artikel
Sprache:	eng
Schlagworte:	Antibiotics. Antiinfectious agents. Antiparasitic agents Antiviral agents Biological and medical sciences Cell Line DEAD Box Protein 58 DEAD-box RNA Helicases - physiology Gastroenterology. Liver. Pancreas. Abdomen Hepacivirus - physiology Hepatitis C virus Host-Pathogen Interactions - physiology Humans Interferon Regulatory Factor-3 - physiology Liver. Biliary tract. Portal circulation. Exocrine pancreas Medical sciences Pharmacology. Drug treatments Receptors, Immunologic RNA, Double-Stranded - physiology RNA, Viral - physiology Signal Transduction - physiology Tumors Virus Replication - physiology
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