Upregulation of EphA3 receptor after spinal cord injury

Spinal cord injury (SCI) releases a cascade of events that leads to the onset of an inhibitory milieu for axonal regeneration. Some of these changes result from the presence of repulsive factors that may restrict axonal outgrowth after trauma. The Eph receptor tyrosine kinase (RTK) family has emerge...

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Veröffentlicht in:	Journal of neurotrauma 2005-08, Vol.22 (8), p.929-935
Hauptverfasser:	IRIZARRY-RAMIREZ, Margarita, WILLSON, Christopher A, CRUZ-ORENGO, Lillian, FIGUEROA, Johnny, VELAZQUEZ, Ixane, JONES, Hope, FOSTER, Roy D, WHITTEMORE, Scott R, MIRANDA, Jorge D
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Anterior Horn Cells - metabolism Astrocytes - metabolism Biological and medical sciences Brain Injuries - genetics Brain Injuries - metabolism Brain Injuries - physiopathology Cell Communication - physiology Disease Models, Animal Female Gene expression Gene Expression Regulation - physiology Genotype & phenotype Glial Fibrillary Acidic Protein - metabolism Growth Cones - metabolism Growth Inhibitors - genetics Growth Inhibitors - metabolism Injuries of the nervous system and the skull. Diseases due to physical agents Kinases Medical sciences Molecular biology Nerve Regeneration - physiology Neural Pathways - metabolism Neural Pathways - physiopathology Neurology Optic Nerve Injuries - genetics Optic Nerve Injuries - metabolism Optic Nerve Injuries - physiopathology Rats Rats, Sprague-Dawley Receptor Protein-Tyrosine Kinases - genetics Receptor Protein-Tyrosine Kinases - metabolism Ribonucleic acid RNA RNA, Messenger - metabolism Spinal Cord - metabolism Spinal Cord - physiopathology Spinal cord injuries Spinal Cord Injuries - genetics Spinal Cord Injuries - metabolism Spinal Cord Injuries - physiopathology Traumas. Diseases due to physical agents Up-Regulation - physiology
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