Mechanism of neuronal injury in cerebral venous thrombosis
The impact of CVT on the brain is wide spectrum, ranging from completely normal parenchyma to brain oedema and/or haemorrhage. Multiple factors relate to neuronal injury in CVT including; dural sinus pressure, increased venous flow velocities, collateralization of venous channels, rate of occlusion,...
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| Veröffentlicht in: | Journal of the Pakistan Medical Association 2006-11, Vol.56 (11), p.509-512 |
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| container_title | Journal of the Pakistan Medical Association |
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| creator | Usman, Uzma Wasay, Mohammad |
| description | The impact of CVT on the brain is wide spectrum, ranging from completely normal parenchyma to brain oedema and/or haemorrhage. Multiple factors relate to neuronal injury in CVT including; dural sinus pressure, increased venous flow velocities, collateralization of venous channels, rate of occlusion, development of cytotoxic and vasogenic oedema, recanalization and accelerated myelination. It is suggested that recanalization of occluded vein, as well as, the presence or absence and the efficiency of intracranial venous collaterals, may have an impact on the extent of brain tissue damage and hence the prognosis of acute CVT. |
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Multiple factors relate to neuronal injury in CVT including; dural sinus pressure, increased venous flow velocities, collateralization of venous channels, rate of occlusion, development of cytotoxic and vasogenic oedema, recanalization and accelerated myelination. 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Multiple factors relate to neuronal injury in CVT including; dural sinus pressure, increased venous flow velocities, collateralization of venous channels, rate of occlusion, development of cytotoxic and vasogenic oedema, recanalization and accelerated myelination. It is suggested that recanalization of occluded vein, as well as, the presence or absence and the efficiency of intracranial venous collaterals, may have an impact on the extent of brain tissue damage and hence the prognosis of acute CVT.</description><subject>Brain Ischemia - etiology</subject><subject>Brain Ischemia - physiopathology</subject><subject>Cerebral Hemorrhage - etiology</subject><subject>Cerebral Hemorrhage - physiopathology</subject><subject>Cerebral Veins - pathology</subject><subject>Demyelinating Diseases - etiology</subject><subject>Humans</subject><subject>Intracranial Thrombosis - complications</subject><subject>Intracranial Thrombosis - drug therapy</subject><subject>Neurons</subject><subject>Prognosis</subject><subject>Risk Factors</subject><subject>Sinus Thrombosis, Intracranial - complications</subject><subject>Sinus Thrombosis, Intracranial - drug therapy</subject><subject>Venous Thrombosis - complications</subject><subject>Venous Thrombosis - drug therapy</subject><issn>0030-9982</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2006</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNo1j01LxDAURbNQnHH0L0hX7govSZu8uJPBLxhxo-uSZF6YDm1ak1aYf2_BcXXgcrjce8HWABJKY1Cs2HXORwChaoArtuKaozQa1-zhnfzBxjb3xRCKSHMaou2KNh7ndFpQeErk0hL9UBzmXEyHNPRuyG2-YZfBdpluz9ywr-enz-1ruft4eds-7sqRi2oqNfDaC82tUt7bSlCNDp2WolImeFRG6wAC9mTJoPcBCHiwgJXFCqV0csPu_3rHNHzPlKemb7OnrrORlkWNQqGhVriId2dxdj3tmzG1vU2n5v-t_AWSsE-r</recordid><startdate>200611</startdate><enddate>200611</enddate><creator>Usman, Uzma</creator><creator>Wasay, Mohammad</creator><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>7X8</scope></search><sort><creationdate>200611</creationdate><title>Mechanism of neuronal injury in cerebral venous thrombosis</title><author>Usman, Uzma ; Wasay, Mohammad</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-p124t-7015c271a66cca42e58b8b732469fc86977f020deae98ccf0e01fa084a84833b3</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2006</creationdate><topic>Brain Ischemia - etiology</topic><topic>Brain Ischemia - physiopathology</topic><topic>Cerebral Hemorrhage - etiology</topic><topic>Cerebral Hemorrhage - physiopathology</topic><topic>Cerebral Veins - pathology</topic><topic>Demyelinating Diseases - etiology</topic><topic>Humans</topic><topic>Intracranial Thrombosis - complications</topic><topic>Intracranial Thrombosis - drug therapy</topic><topic>Neurons</topic><topic>Prognosis</topic><topic>Risk Factors</topic><topic>Sinus Thrombosis, Intracranial - complications</topic><topic>Sinus Thrombosis, Intracranial - drug therapy</topic><topic>Venous Thrombosis - complications</topic><topic>Venous Thrombosis - drug therapy</topic><toplevel>online_resources</toplevel><creatorcontrib>Usman, Uzma</creatorcontrib><creatorcontrib>Wasay, Mohammad</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>MEDLINE - Academic</collection><jtitle>Journal of the Pakistan Medical Association</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Usman, Uzma</au><au>Wasay, Mohammad</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Mechanism of neuronal injury in cerebral venous thrombosis</atitle><jtitle>Journal of the Pakistan Medical Association</jtitle><addtitle>J Pak Med Assoc</addtitle><date>2006-11</date><risdate>2006</risdate><volume>56</volume><issue>11</issue><spage>509</spage><epage>512</epage><pages>509-512</pages><issn>0030-9982</issn><abstract>The impact of CVT on the brain is wide spectrum, ranging from completely normal parenchyma to brain oedema and/or haemorrhage. Multiple factors relate to neuronal injury in CVT including; dural sinus pressure, increased venous flow velocities, collateralization of venous channels, rate of occlusion, development of cytotoxic and vasogenic oedema, recanalization and accelerated myelination. It is suggested that recanalization of occluded vein, as well as, the presence or absence and the efficiency of intracranial venous collaterals, may have an impact on the extent of brain tissue damage and hence the prognosis of acute CVT.</abstract><cop>Pakistan</cop><pmid>17183978</pmid><tpages>4</tpages></addata></record> |
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| ispartof | Journal of the Pakistan Medical Association, 2006-11, Vol.56 (11), p.509-512 |
| issn | 0030-9982 |
| language | eng |
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| source | MEDLINE; DOAJ Directory of Open Access Journals; EZB Electronic Journals Library |
| subjects | Brain Ischemia - etiology Brain Ischemia - physiopathology Cerebral Hemorrhage - etiology Cerebral Hemorrhage - physiopathology Cerebral Veins - pathology Demyelinating Diseases - etiology Humans Intracranial Thrombosis - complications Intracranial Thrombosis - drug therapy Neurons Prognosis Risk Factors Sinus Thrombosis, Intracranial - complications Sinus Thrombosis, Intracranial - drug therapy Venous Thrombosis - complications Venous Thrombosis - drug therapy |
| title | Mechanism of neuronal injury in cerebral venous thrombosis |
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