Investigation of the functional variant c.-169T > C of the Fc receptor-like 3 (FCRL3) gene in alopecia areata

Investigation of the functional variant c.-169T > C of the Fc receptor-like 3 (FCRL3) gene in alopecia areata

A functional variant in the Fc receptor‐like 3 (FCRL3) gene has been implicated in susceptibility to autoimmune diseases such as rheumatoid arthritis, systemic lupus erythematosus and autoimmune thyroid disease. Investigating a large case‐control sample of patients with alopecia areata (AA), we foun...

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Veröffentlicht in:International journal of immunogenetics 2006-12, Vol.33 (6), p.393-395
Hauptverfasser: Schäfer, N., Blaumeiser, B., Becker, T., Freudenberg-Hua, Y., Hanneken, S., Eigelshoven, S., Schmael, C., Lambert, J., De Weert, J., Kruse, R., Nöthen, M. M., Betz, R. C.
Format: Artikel
Sprache:eng
Schlagworte:
Adolescent
Adult
Aged
Alopecia Areata - genetics
Case-Control Studies
Child
Cytosine
Female
Genetic Predisposition to Disease
Genetic Variation
Humans
Male
Middle Aged
Polymorphism, Single Nucleotide
Receptors, Immunologic - genetics
Thymine
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Zusammenfassung:A functional variant in the Fc receptor‐like 3 (FCRL3) gene has been implicated in susceptibility to autoimmune diseases such as rheumatoid arthritis, systemic lupus erythematosus and autoimmune thyroid disease. Investigating a large case‐control sample of patients with alopecia areata (AA), we found no evidence for the involvement of FCRL3 in susceptibility to AA.
ISSN:1744-3121
1744-313X
DOI:10.1111/j.1744-313X.2006.00633.x