Inhaled nitric oxide decreases infarction size and improves left ventricular function in a murine model of myocardial ischemia-reperfusion injury

1 Department of Anesthesia and Critical Care, 2 Cardiovascular Research Center, 3 Cardiac Ultrasound Laboratory, Division of Cardiology, Department of Medicine, and 4 Department of Pathology, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts; and 5 Cardiovascular Branc...

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Veröffentlicht in:	American journal of physiology. Heart and circulatory physiology 2006-07, Vol.291 (1), p.H379-H384
Hauptverfasser:	Hataishi, Ryuji, Rodrigues, Ana Clara, Neilan, Tomas G, Morgan, John G, Buys, Emmanuel, Shiva, Sruti, Tambouret, Rosemary, Jassal, Davinder S, Raher, Michael J, Furutani, Elissa, Ichinose, Fumito, Gladwin, Mark T, Rosenzweig, Anthony, Zapol, Warren M, Picard, Michael H, Bloch, Kenneth D, Scherrer-Crosbie, Marielle
Format:	Artikel
Sprache:	eng
Schlagworte:	Administration, Inhalation Animals Disease Models, Animal Male Mice Mice, Inbred C57BL Myocardial Reperfusion Injury - complications Myocardial Reperfusion Injury - pathology Myocardial Reperfusion Injury - physiopathology Myocardial Reperfusion Injury - prevention & control Nitric Oxide - administration & dosage Severity of Illness Index Treatment Outcome Ventricular Dysfunction, Left - etiology Ventricular Dysfunction, Left - pathology Ventricular Dysfunction, Left - physiopathology Ventricular Dysfunction, Left - prevention & control
Online-Zugang:	Volltext
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