PGE in the regulation of programmed erythrocyte death

Hyperosmotic shock, energy depletion, or removal of extracellular Cl(-) activates Ca(2+)-permeable cation channels in erythrocyte membranes. Subsequent Ca(2+) entry induces erythrocyte shrinkage and exposure of phosphatidylserine (PS) at the erythrocyte surface. PS-exposing cells are engulfed by mac...

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Veröffentlicht in:	Cell death and differentiation 2005-05, Vol.12 (5), p.415-428
Hauptverfasser:	Lang, P A, Kempe, D S, Myssina, S, Tanneur, V, Birka, C, Laufer, S, Lang, F, Wieder, T, Huber, S M
Format:	Artikel
Sprache:	eng
Schlagworte:	Ankyrins - metabolism Annexins - metabolism Apoptosis - drug effects Calcium - metabolism Calcium Channels - drug effects Calpain - metabolism Cell Size - drug effects Chlorides - metabolism Cyclooxygenase Inhibitors - pharmacology Cytosol - drug effects Diclofenac - pharmacology Enzyme Inhibitors - pharmacology Erythrocytes - drug effects Flow Cytometry Humans Models, Biological Osmotic Pressure - drug effects Patch-Clamp Techniques Phosphatidylserines - metabolism Phospholipases A - metabolism Prostaglandins E - pharmacology Prostaglandins E - secretion Quinacrine - pharmacology Saline Solution, Hypertonic
Online-Zugang:	Volltext
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