Are type II endoleaks after endovascular aneurysm repair endograft dependent?
Most clinical end points after endovascular aneurysm repair (EVAR) are endograft-specific, but type II endoleaks have been assumed to be an unavoidable consequence of the repair method and independent of the type of endograft used. Some recent data have suggested that the rate of type II endoleaks m...
Gespeichert in:
| Veröffentlicht in: | Journal of vascular surgery 2006-04, Vol.43 (4), p.657-661 |
|---|---|
| Hauptverfasser: | , , , , , , , , , |
| Format: | Artikel |
| Sprache: | eng |
| Schlagworte: | |
| Online-Zugang: | Volltext |
| Tags: |
Tag hinzufügen
Keine Tags, Fügen Sie den ersten Tag hinzu!
|
| container_end_page | 661 |
|---|---|
| container_issue | 4 |
| container_start_page | 657 |
| container_title | Journal of vascular surgery |
| container_volume | 43 |
| creator | Sheehan, Maureen K. Ouriel, Kenneth Greenberg, Roy McCann, Richard Murphy, Michael Fillinger, Mark Wyers, Mark Carpenter, Jeffrey Fairman, Ronald Makaroun, Michel S. |
| description | Most clinical end points after endovascular aneurysm repair (EVAR) are endograft-specific, but type II endoleaks have been assumed to be an unavoidable consequence of the repair method and independent of the type of endograft used. Some recent data have suggested that the rate of type II endoleaks may also be graft-dependent. We reviewed a large clinical experience with six endografts to determine the behavior of type II endoleaks and whether they are graft-specific.
All elective EVAR cases from five university institutions from 1996 to 2003 were retrospectively analyzed. Endografts used in |
| doi_str_mv | 10.1016/j.jvs.2005.12.044 |
| format | Article |
| fullrecord | <record><control><sourceid>proquest_cross</sourceid><recordid>TN_cdi_proquest_miscellaneous_67859048</recordid><sourceformat>XML</sourceformat><sourcesystem>PC</sourcesystem><els_id>S0741521406000073</els_id><sourcerecordid>67859048</sourcerecordid><originalsourceid>FETCH-LOGICAL-c424t-bd65666efa90a08a068cde19ee002c07e3669eff763be9446fe4be9cba4965b43</originalsourceid><addsrcrecordid>eNp9kE1Lw0AQhhdRtH78AC-Si94SZ9LNJMGDlOJHQfGi52WzmUhqmsTdpNB_79YWvHmaGeaZ4eUR4hIhQkC6XUbLtYtigCTCOAIpD8QEIU9DyiA_FBNIJYZJjPJEnDq3BEBMsvRYnCARUow0Ea8zy8Gw6TlYLAJuy65h_eUCXQ1sf-e1dmZstA10y6PduFVgudf1bvlpPRiU3PuB2-H-XBxVunF8sa9n4uPx4X3-HL68PS3ms5fQyFgOYVFSQkRc6Rw0ZBooMyVjzgwQG0h5SpRzVaU0LTiXkiqWvjGFljklhZyeiZvd39523yO7Qa1qZ7hpfMhudIrSLMlBZh7EHWhs55zlSvW2Xmm7UQhq61AtlXeotg4Vxso79DdX--djseLy72IvzQPXe8C70U1ldWtq98elKWSI6Lm7Hcdexbpmq5ypuTVc1pbNoMqu_ifGDzqAj5c</addsrcrecordid><sourcetype>Aggregation Database</sourcetype><iscdi>true</iscdi><recordtype>article</recordtype><pqid>67859048</pqid></control><display><type>article</type><title>Are type II endoleaks after endovascular aneurysm repair endograft dependent?</title><source>MEDLINE</source><source>Elsevier ScienceDirect Journals Complete</source><source>EZB-FREE-00999 freely available EZB journals</source><creator>Sheehan, Maureen K. ; Ouriel, Kenneth ; Greenberg, Roy ; McCann, Richard ; Murphy, Michael ; Fillinger, Mark ; Wyers, Mark ; Carpenter, Jeffrey ; Fairman, Ronald ; Makaroun, Michel S.</creator><creatorcontrib>Sheehan, Maureen K. ; Ouriel, Kenneth ; Greenberg, Roy ; McCann, Richard ; Murphy, Michael ; Fillinger, Mark ; Wyers, Mark ; Carpenter, Jeffrey ; Fairman, Ronald ; Makaroun, Michel S.</creatorcontrib><description>Most clinical end points after endovascular aneurysm repair (EVAR) are endograft-specific, but type II endoleaks have been assumed to be an unavoidable consequence of the repair method and independent of the type of endograft used. Some recent data have suggested that the rate of type II endoleaks may also be graft-dependent. We reviewed a large clinical experience with six endografts to determine the behavior of type II endoleaks and whether they are graft-specific.
All elective EVAR cases from five university institutions from 1996 to 2003 were retrospectively analyzed. Endografts used in <50 patients were excluded. Endoleaks were diagnosed and classified from contrast-enhanced computed tomography (CT) scans by the treating surgeons. Results of angiography and interventions for endoleaks were tracked. The rate of type II endoleaks was compared among endografts at 1, 6, and 12 months, and yearly thereafter. Statistical significance was defined as P < .05.
During the study period, 1909 patients underwent elective EVAR and had an adequate imaging follow-up at one of the specified time points. At 1 month, the overall rate of type II endoleak was 14.0% (range, 9.8% to 25.2%.) The Excluder had a significantly higher incidence of type II endoleaks at 1 month but was similar to most other grafts during longer follow-up. At 6 months, the overall rate of type II endoleak was 16.3% (range, 8.3% to 16.8%). The Talent and Lifepath had an apparent lower initial rate of type II leaks, but this was only significant for the Talent at 6 months compared with Excluder, Zenith, and Ancure, and at 1 year compared with Excluder and Zenith. No graft had a long-term statistically significant difference in the rate of type II endoleak formation. Intervention rates varied by institution and graft type but in general were quite low. Of 25 successful interventions (Ancure, 12; AneuRx, 8; Excluder, 2; Lifepath, 2; Zenith, 1; Talent, 0), 21 were performed during the first year. Interim spontaneous resolution, defined as a negative CT scan after a CT positive for endoleak, was high, especially in the first year. Resolution of type II endoleaks occurred in 54 (33%) of 164 between 1 and 6 months, in 37 (33%) of 112 between 6 and 12 months, in 20 (35%) of 57 from 12 to 24 months, and in 5 (20%) of 25 between 24 and 36 months. The various grafts had a nearly identical pattern, but the rates were highest for the Talent. Late appearing endoleaks, defined as a positive CT after a negative CT, were frequent. At 6 months, 44 (30%) of 147 type II endoleaks were newly diagnosed. The rates were 37 (35%) of 107 at 12 months, 15 (27%) of 56 at 2 years and 5 (25%) of 20 at 3 years. No conversions to open repair for type II endoleaks were noted in the first 4 years. The thrombus burden could not be determined in this analysis.
Type II endoleaks occur in nearly 15% of patients treated by EVAR. The early incidence varies only slightly with graft type. The long-term prevalence and clinical significance are masked by different treatment patterns, spontaneous resolution, newly evident endoleaks, and aneurysm size at initial treatment.</description><identifier>ISSN: 0741-5214</identifier><identifier>EISSN: 1097-6809</identifier><identifier>DOI: 10.1016/j.jvs.2005.12.044</identifier><identifier>PMID: 16616216</identifier><identifier>CODEN: JVSUES</identifier><language>eng</language><publisher>New York, NY: Elsevier Inc</publisher><subject>Adult ; Aged ; Aortic Aneurysm, Abdominal - diagnostic imaging ; Aortic Aneurysm, Abdominal - surgery ; Aortography ; Biological and medical sciences ; Blood and lymphatic vessels ; Blood Vessel Prosthesis ; Blood Vessel Prosthesis Implantation - adverse effects ; Blood Vessel Prosthesis Implantation - methods ; Cardiology. Vascular system ; Diseases of the aorta ; Female ; Follow-Up Studies ; Humans ; Incidence ; Male ; Medical sciences ; Middle Aged ; Postoperative Complications - epidemiology ; Postoperative Complications - pathology ; Probability ; Prosthesis Design ; Prosthesis Failure ; Retrospective Studies ; Risk Assessment ; Sarcoidosis. Granulomatous diseases of unproved etiology. Connective tissue diseases. Elastic tissue diseases. Vasculitis ; Surgery (general aspects). Transplantations, organ and tissue grafts. Graft diseases ; Time Factors ; Tomography, X-Ray Computed ; Vascular surgery: aorta, extremities, vena cava. Surgery of the lymphatic vessels</subject><ispartof>Journal of vascular surgery, 2006-04, Vol.43 (4), p.657-661</ispartof><rights>2006 The Society for Vascular Surgery</rights><rights>2006 INIST-CNRS</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c424t-bd65666efa90a08a068cde19ee002c07e3669eff763be9446fe4be9cba4965b43</citedby><cites>FETCH-LOGICAL-c424t-bd65666efa90a08a068cde19ee002c07e3669eff763be9446fe4be9cba4965b43</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktohtml>$$Uhttps://dx.doi.org/10.1016/j.jvs.2005.12.044$$EHTML$$P50$$Gelsevier$$Hfree_for_read</linktohtml><link.rule.ids>314,780,784,3550,27924,27925,45995</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=17708111$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/16616216$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Sheehan, Maureen K.</creatorcontrib><creatorcontrib>Ouriel, Kenneth</creatorcontrib><creatorcontrib>Greenberg, Roy</creatorcontrib><creatorcontrib>McCann, Richard</creatorcontrib><creatorcontrib>Murphy, Michael</creatorcontrib><creatorcontrib>Fillinger, Mark</creatorcontrib><creatorcontrib>Wyers, Mark</creatorcontrib><creatorcontrib>Carpenter, Jeffrey</creatorcontrib><creatorcontrib>Fairman, Ronald</creatorcontrib><creatorcontrib>Makaroun, Michel S.</creatorcontrib><title>Are type II endoleaks after endovascular aneurysm repair endograft dependent?</title><title>Journal of vascular surgery</title><addtitle>J Vasc Surg</addtitle><description>Most clinical end points after endovascular aneurysm repair (EVAR) are endograft-specific, but type II endoleaks have been assumed to be an unavoidable consequence of the repair method and independent of the type of endograft used. Some recent data have suggested that the rate of type II endoleaks may also be graft-dependent. We reviewed a large clinical experience with six endografts to determine the behavior of type II endoleaks and whether they are graft-specific.
All elective EVAR cases from five university institutions from 1996 to 2003 were retrospectively analyzed. Endografts used in <50 patients were excluded. Endoleaks were diagnosed and classified from contrast-enhanced computed tomography (CT) scans by the treating surgeons. Results of angiography and interventions for endoleaks were tracked. The rate of type II endoleaks was compared among endografts at 1, 6, and 12 months, and yearly thereafter. Statistical significance was defined as P < .05.
During the study period, 1909 patients underwent elective EVAR and had an adequate imaging follow-up at one of the specified time points. At 1 month, the overall rate of type II endoleak was 14.0% (range, 9.8% to 25.2%.) The Excluder had a significantly higher incidence of type II endoleaks at 1 month but was similar to most other grafts during longer follow-up. At 6 months, the overall rate of type II endoleak was 16.3% (range, 8.3% to 16.8%). The Talent and Lifepath had an apparent lower initial rate of type II leaks, but this was only significant for the Talent at 6 months compared with Excluder, Zenith, and Ancure, and at 1 year compared with Excluder and Zenith. No graft had a long-term statistically significant difference in the rate of type II endoleak formation. Intervention rates varied by institution and graft type but in general were quite low. Of 25 successful interventions (Ancure, 12; AneuRx, 8; Excluder, 2; Lifepath, 2; Zenith, 1; Talent, 0), 21 were performed during the first year. Interim spontaneous resolution, defined as a negative CT scan after a CT positive for endoleak, was high, especially in the first year. Resolution of type II endoleaks occurred in 54 (33%) of 164 between 1 and 6 months, in 37 (33%) of 112 between 6 and 12 months, in 20 (35%) of 57 from 12 to 24 months, and in 5 (20%) of 25 between 24 and 36 months. The various grafts had a nearly identical pattern, but the rates were highest for the Talent. Late appearing endoleaks, defined as a positive CT after a negative CT, were frequent. At 6 months, 44 (30%) of 147 type II endoleaks were newly diagnosed. The rates were 37 (35%) of 107 at 12 months, 15 (27%) of 56 at 2 years and 5 (25%) of 20 at 3 years. No conversions to open repair for type II endoleaks were noted in the first 4 years. The thrombus burden could not be determined in this analysis.
Type II endoleaks occur in nearly 15% of patients treated by EVAR. The early incidence varies only slightly with graft type. The long-term prevalence and clinical significance are masked by different treatment patterns, spontaneous resolution, newly evident endoleaks, and aneurysm size at initial treatment.</description><subject>Adult</subject><subject>Aged</subject><subject>Aortic Aneurysm, Abdominal - diagnostic imaging</subject><subject>Aortic Aneurysm, Abdominal - surgery</subject><subject>Aortography</subject><subject>Biological and medical sciences</subject><subject>Blood and lymphatic vessels</subject><subject>Blood Vessel Prosthesis</subject><subject>Blood Vessel Prosthesis Implantation - adverse effects</subject><subject>Blood Vessel Prosthesis Implantation - methods</subject><subject>Cardiology. Vascular system</subject><subject>Diseases of the aorta</subject><subject>Female</subject><subject>Follow-Up Studies</subject><subject>Humans</subject><subject>Incidence</subject><subject>Male</subject><subject>Medical sciences</subject><subject>Middle Aged</subject><subject>Postoperative Complications - epidemiology</subject><subject>Postoperative Complications - pathology</subject><subject>Probability</subject><subject>Prosthesis Design</subject><subject>Prosthesis Failure</subject><subject>Retrospective Studies</subject><subject>Risk Assessment</subject><subject>Sarcoidosis. Granulomatous diseases of unproved etiology. Connective tissue diseases. Elastic tissue diseases. Vasculitis</subject><subject>Surgery (general aspects). Transplantations, organ and tissue grafts. Graft diseases</subject><subject>Time Factors</subject><subject>Tomography, X-Ray Computed</subject><subject>Vascular surgery: aorta, extremities, vena cava. Surgery of the lymphatic vessels</subject><issn>0741-5214</issn><issn>1097-6809</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2006</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNp9kE1Lw0AQhhdRtH78AC-Si94SZ9LNJMGDlOJHQfGi52WzmUhqmsTdpNB_79YWvHmaGeaZ4eUR4hIhQkC6XUbLtYtigCTCOAIpD8QEIU9DyiA_FBNIJYZJjPJEnDq3BEBMsvRYnCARUow0Ea8zy8Gw6TlYLAJuy65h_eUCXQ1sf-e1dmZstA10y6PduFVgudf1bvlpPRiU3PuB2-H-XBxVunF8sa9n4uPx4X3-HL68PS3ms5fQyFgOYVFSQkRc6Rw0ZBooMyVjzgwQG0h5SpRzVaU0LTiXkiqWvjGFljklhZyeiZvd39523yO7Qa1qZ7hpfMhudIrSLMlBZh7EHWhs55zlSvW2Xmm7UQhq61AtlXeotg4Vxso79DdX--djseLy72IvzQPXe8C70U1ldWtq98elKWSI6Lm7Hcdexbpmq5ypuTVc1pbNoMqu_ifGDzqAj5c</recordid><startdate>20060401</startdate><enddate>20060401</enddate><creator>Sheehan, Maureen K.</creator><creator>Ouriel, Kenneth</creator><creator>Greenberg, Roy</creator><creator>McCann, Richard</creator><creator>Murphy, Michael</creator><creator>Fillinger, Mark</creator><creator>Wyers, Mark</creator><creator>Carpenter, Jeffrey</creator><creator>Fairman, Ronald</creator><creator>Makaroun, Michel S.</creator><general>Elsevier Inc</general><general>Elsevier</general><scope>6I.</scope><scope>AAFTH</scope><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>20060401</creationdate><title>Are type II endoleaks after endovascular aneurysm repair endograft dependent?</title><author>Sheehan, Maureen K. ; Ouriel, Kenneth ; Greenberg, Roy ; McCann, Richard ; Murphy, Michael ; Fillinger, Mark ; Wyers, Mark ; Carpenter, Jeffrey ; Fairman, Ronald ; Makaroun, Michel S.</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c424t-bd65666efa90a08a068cde19ee002c07e3669eff763be9446fe4be9cba4965b43</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2006</creationdate><topic>Adult</topic><topic>Aged</topic><topic>Aortic Aneurysm, Abdominal - diagnostic imaging</topic><topic>Aortic Aneurysm, Abdominal - surgery</topic><topic>Aortography</topic><topic>Biological and medical sciences</topic><topic>Blood and lymphatic vessels</topic><topic>Blood Vessel Prosthesis</topic><topic>Blood Vessel Prosthesis Implantation - adverse effects</topic><topic>Blood Vessel Prosthesis Implantation - methods</topic><topic>Cardiology. Vascular system</topic><topic>Diseases of the aorta</topic><topic>Female</topic><topic>Follow-Up Studies</topic><topic>Humans</topic><topic>Incidence</topic><topic>Male</topic><topic>Medical sciences</topic><topic>Middle Aged</topic><topic>Postoperative Complications - epidemiology</topic><topic>Postoperative Complications - pathology</topic><topic>Probability</topic><topic>Prosthesis Design</topic><topic>Prosthesis Failure</topic><topic>Retrospective Studies</topic><topic>Risk Assessment</topic><topic>Sarcoidosis. Granulomatous diseases of unproved etiology. Connective tissue diseases. Elastic tissue diseases. Vasculitis</topic><topic>Surgery (general aspects). Transplantations, organ and tissue grafts. Graft diseases</topic><topic>Time Factors</topic><topic>Tomography, X-Ray Computed</topic><topic>Vascular surgery: aorta, extremities, vena cava. Surgery of the lymphatic vessels</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Sheehan, Maureen K.</creatorcontrib><creatorcontrib>Ouriel, Kenneth</creatorcontrib><creatorcontrib>Greenberg, Roy</creatorcontrib><creatorcontrib>McCann, Richard</creatorcontrib><creatorcontrib>Murphy, Michael</creatorcontrib><creatorcontrib>Fillinger, Mark</creatorcontrib><creatorcontrib>Wyers, Mark</creatorcontrib><creatorcontrib>Carpenter, Jeffrey</creatorcontrib><creatorcontrib>Fairman, Ronald</creatorcontrib><creatorcontrib>Makaroun, Michel S.</creatorcontrib><collection>ScienceDirect Open Access Titles</collection><collection>Elsevier:ScienceDirect:Open Access</collection><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Journal of vascular surgery</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Sheehan, Maureen K.</au><au>Ouriel, Kenneth</au><au>Greenberg, Roy</au><au>McCann, Richard</au><au>Murphy, Michael</au><au>Fillinger, Mark</au><au>Wyers, Mark</au><au>Carpenter, Jeffrey</au><au>Fairman, Ronald</au><au>Makaroun, Michel S.</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Are type II endoleaks after endovascular aneurysm repair endograft dependent?</atitle><jtitle>Journal of vascular surgery</jtitle><addtitle>J Vasc Surg</addtitle><date>2006-04-01</date><risdate>2006</risdate><volume>43</volume><issue>4</issue><spage>657</spage><epage>661</epage><pages>657-661</pages><issn>0741-5214</issn><eissn>1097-6809</eissn><coden>JVSUES</coden><abstract>Most clinical end points after endovascular aneurysm repair (EVAR) are endograft-specific, but type II endoleaks have been assumed to be an unavoidable consequence of the repair method and independent of the type of endograft used. Some recent data have suggested that the rate of type II endoleaks may also be graft-dependent. We reviewed a large clinical experience with six endografts to determine the behavior of type II endoleaks and whether they are graft-specific.
All elective EVAR cases from five university institutions from 1996 to 2003 were retrospectively analyzed. Endografts used in <50 patients were excluded. Endoleaks were diagnosed and classified from contrast-enhanced computed tomography (CT) scans by the treating surgeons. Results of angiography and interventions for endoleaks were tracked. The rate of type II endoleaks was compared among endografts at 1, 6, and 12 months, and yearly thereafter. Statistical significance was defined as P < .05.
During the study period, 1909 patients underwent elective EVAR and had an adequate imaging follow-up at one of the specified time points. At 1 month, the overall rate of type II endoleak was 14.0% (range, 9.8% to 25.2%.) The Excluder had a significantly higher incidence of type II endoleaks at 1 month but was similar to most other grafts during longer follow-up. At 6 months, the overall rate of type II endoleak was 16.3% (range, 8.3% to 16.8%). The Talent and Lifepath had an apparent lower initial rate of type II leaks, but this was only significant for the Talent at 6 months compared with Excluder, Zenith, and Ancure, and at 1 year compared with Excluder and Zenith. No graft had a long-term statistically significant difference in the rate of type II endoleak formation. Intervention rates varied by institution and graft type but in general were quite low. Of 25 successful interventions (Ancure, 12; AneuRx, 8; Excluder, 2; Lifepath, 2; Zenith, 1; Talent, 0), 21 were performed during the first year. Interim spontaneous resolution, defined as a negative CT scan after a CT positive for endoleak, was high, especially in the first year. Resolution of type II endoleaks occurred in 54 (33%) of 164 between 1 and 6 months, in 37 (33%) of 112 between 6 and 12 months, in 20 (35%) of 57 from 12 to 24 months, and in 5 (20%) of 25 between 24 and 36 months. The various grafts had a nearly identical pattern, but the rates were highest for the Talent. Late appearing endoleaks, defined as a positive CT after a negative CT, were frequent. At 6 months, 44 (30%) of 147 type II endoleaks were newly diagnosed. The rates were 37 (35%) of 107 at 12 months, 15 (27%) of 56 at 2 years and 5 (25%) of 20 at 3 years. No conversions to open repair for type II endoleaks were noted in the first 4 years. The thrombus burden could not be determined in this analysis.
Type II endoleaks occur in nearly 15% of patients treated by EVAR. The early incidence varies only slightly with graft type. The long-term prevalence and clinical significance are masked by different treatment patterns, spontaneous resolution, newly evident endoleaks, and aneurysm size at initial treatment.</abstract><cop>New York, NY</cop><pub>Elsevier Inc</pub><pmid>16616216</pmid><doi>10.1016/j.jvs.2005.12.044</doi><tpages>5</tpages><oa>free_for_read</oa></addata></record> |
| fulltext | fulltext |
| identifier | ISSN: 0741-5214 |
| ispartof | Journal of vascular surgery, 2006-04, Vol.43 (4), p.657-661 |
| issn | 0741-5214 1097-6809 |
| language | eng |
| recordid | cdi_proquest_miscellaneous_67859048 |
| source | MEDLINE; Elsevier ScienceDirect Journals Complete; EZB-FREE-00999 freely available EZB journals |
| subjects | Adult Aged Aortic Aneurysm, Abdominal - diagnostic imaging Aortic Aneurysm, Abdominal - surgery Aortography Biological and medical sciences Blood and lymphatic vessels Blood Vessel Prosthesis Blood Vessel Prosthesis Implantation - adverse effects Blood Vessel Prosthesis Implantation - methods Cardiology. Vascular system Diseases of the aorta Female Follow-Up Studies Humans Incidence Male Medical sciences Middle Aged Postoperative Complications - epidemiology Postoperative Complications - pathology Probability Prosthesis Design Prosthesis Failure Retrospective Studies Risk Assessment Sarcoidosis. Granulomatous diseases of unproved etiology. Connective tissue diseases. Elastic tissue diseases. Vasculitis Surgery (general aspects). Transplantations, organ and tissue grafts. Graft diseases Time Factors Tomography, X-Ray Computed Vascular surgery: aorta, extremities, vena cava. Surgery of the lymphatic vessels |
| title | Are type II endoleaks after endovascular aneurysm repair endograft dependent? |
| url | https://sfx.bib-bvb.de/sfx_tum?ctx_ver=Z39.88-2004&ctx_enc=info:ofi/enc:UTF-8&ctx_tim=2024-12-24T08%3A01%3A14IST&url_ver=Z39.88-2004&url_ctx_fmt=infofi/fmt:kev:mtx:ctx&rfr_id=info:sid/primo.exlibrisgroup.com:primo3-Article-proquest_cross&rft_val_fmt=info:ofi/fmt:kev:mtx:journal&rft.genre=article&rft.atitle=Are%20type%20II%20endoleaks%20after%20endovascular%20aneurysm%20repair%20endograft%20dependent?&rft.jtitle=Journal%20of%20vascular%20surgery&rft.au=Sheehan,%20Maureen%20K.&rft.date=2006-04-01&rft.volume=43&rft.issue=4&rft.spage=657&rft.epage=661&rft.pages=657-661&rft.issn=0741-5214&rft.eissn=1097-6809&rft.coden=JVSUES&rft_id=info:doi/10.1016/j.jvs.2005.12.044&rft_dat=%3Cproquest_cross%3E67859048%3C/proquest_cross%3E%3Curl%3E%3C/url%3E&disable_directlink=true&sfx.directlink=off&sfx.report_link=0&rft_id=info:oai/&rft_pqid=67859048&rft_id=info:pmid/16616216&rft_els_id=S0741521406000073&rfr_iscdi=true |