Receptor for advanced-glycation end products : Key modulator of myocardial ischemic injury

The beneficial effects of reperfusion therapies have been limited by the amount of ischemic damage that occurs before reperfusion. To enable development of interventions to reduce cell injury, our research has focused on understanding mechanisms involved in cardiac cell death after ischemia/reperfus...

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Veröffentlicht in:	Circulation (New York, N.Y.) N.Y.), 2006-03, Vol.113 (9), p.1226-1234
Hauptverfasser:	BUCCIARELLI, Loredana G, KANEKO, Michiyo, FEI SONG, WU QU, GOMEZ, Teodoro, YU SHAN ZOU, SHI FANG YAN, SCHMIDT, Ann Marie, RAMASAMY, Ravichandran, ANANTHAKRISHNAN, Radha, HARJA, Evis, LEE, Larisse K, HWANG, Yuying C, LERNER, Shulamit, BAKR, Soliman, QING LI, YAN LU
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Antihypertensive agents Biological and medical sciences Blood and lymphatic vessels Cardiology. Vascular system Cardiovascular system Coronary heart disease Cyclic GMP - analysis Diseases of the peripheral vessels. Diseases of the vena cava. Miscellaneous Energy Metabolism Heart Male Medical sciences Mice Mice, Knockout Myocardial Ischemia - metabolism Myocardial Ischemia - pathology Myocardial Reperfusion Injury - metabolism Myocardial Reperfusion Injury - pathology Nitric Oxide - analysis Nitric Oxide Synthase Type II - analysis Pharmacology. Drug treatments Rats Receptor for Advanced Glycation End Products Receptors, Immunologic - deficiency Receptors, Immunologic - physiology Tyrosine - analogs & derivatives Tyrosine - analysis Up-Regulation
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