Temporal analysis of signaling pathways activated in a murine model of two-kidney, one-clip hypertension

1 Department of Experimental Pathology, ; 2 Department of Transplantation Biology, ; 3 Department of Nephrology and Hypertension, and ; 4 Department of Immunology, Mayo Clinic College of Medicine, Rochester, Minnesota Submitted July 25, 2008 ; accepted in final form July 16, 2009 Unilateral renal ar...

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Veröffentlicht in:	American journal of physiology. Renal physiology 2009-10, Vol.297 (4), p.F1055-F1068
Hauptverfasser:	Cheng, Jingfei, Zhou, Wei, Warner, Gina M, Knudsen, Bruce E, Garovic, Vesna D, Gray, Catherine E, Lerman, Lilach O, Platt, Jeffrey L, Romero, J. Carlos, Textor, Stephen C, Nath, Karl A, Grande, Joseph P
Format:	Artikel
Sprache:	eng
Schlagworte:	Actins - metabolism Animals Apoptosis Atrophy Cell cycle Cell growth Cell Proliferation Chemokine CCL2 - metabolism Collagen - metabolism Cyclin-Dependent Kinase Inhibitor p21 - metabolism Cyclin-Dependent Kinase Inhibitor p27 - metabolism Cyclins - metabolism Extracellular Signal-Regulated MAP Kinases - metabolism Fibrosis Gene expression Hyperplasia Hypertension, Renovascular - genetics Hypertension, Renovascular - metabolism Hypertension, Renovascular - pathology Hypertrophy Interphase Kidney - pathology Kidney diseases Male Mice Mice, Inbred C57BL Proteins Rodents Signal Transduction Studies Time Factors Transforming Growth Factor beta1 - metabolism Tumor Suppressor Protein p53 - metabolism
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