Xanthine oxidase inhibitors improve energetics and function after infarction in failing mouse hearts

1 Department of Radiology, Division of Magnetic Resonance Research, and 2 Department of Pediatrics, the Johns Hopkins University School of Medicine, Baltimore, Maryland; and 3 Department of Medicine, Division of Cardiology, the Johns Hopkins Hospital, Baltimore, Maryland Submitted 4 August 2005 ; ac...

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Veröffentlicht in:	American journal of physiology. Heart and circulatory physiology 2006-02, Vol.290 (2), p.H837-H843
Hauptverfasser:	Naumova, Anna V, Chacko, Vadappuram P, Ouwerkerk, Ronald, Stull, Linda, Marban, Eduardo, Weiss, Robert G
Format:	Artikel
Sprache:	eng
Schlagworte:	Adenosine Triphosphate - biosynthesis Allopurinol - pharmacology Animals Cardiac Output, Low - complications Energy Metabolism - drug effects Enzyme Inhibitors - pharmacology Heart - drug effects Heart - physiopathology Magnetic Resonance Imaging Magnetic Resonance Spectroscopy Mice Myocardial Infarction - diagnosis Myocardial Infarction - etiology Myocardial Infarction - metabolism Myocardial Infarction - physiopathology Myocardium - metabolism Oxypurinol - pharmacology Phosphocreatine - metabolism Stroke Volume - drug effects Ventricular Function - drug effects Ventricular Remodeling - drug effects Xanthine Oxidase - antagonists & inhibitors
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