Effect of indomethacin on E-cadherin and β -catenin expression in HT-29 colon cancer cells

Nonsteroidal anti-inflammatory drugs (NSAIDs) lower the incidence of and mortality from colon cancer. Although there is much evidence from epidemiological and laboratory studies that NSAIDs have antitumor activity and reduce the incidence of colon cancer, the mechanism of action remains unknown. In...

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Veröffentlicht in:	Experimental and molecular pathology 2006-02, Vol.80 (1), p.91-96
Hauptverfasser:	Kapitanović, Sanja, Čačev, Tamara, Antica, Mariastefania, Kralj, Marijeta, Cavrić, Gordana, Pavelić, Krešimir, Spaventi, Radan
Format:	Artikel
Sprache:	eng
Schlagworte:	Active Transport, Cell Nucleus Adaptor Proteins, Vesicular Transport - metabolism Anti-Inflammatory Agents, Non-Steroidal - pharmacology Antineoplastic Agents - pharmacology APC Apoptosis beta Catenin - metabolism Biological and medical sciences Cadherins - metabolism Cell Membrane - metabolism Cell Nucleus - metabolism Cell Proliferation Colon carcinoma Colonic Neoplasms - drug therapy Colonic Neoplasms - metabolism COX-2 Cyclooxygenase 2 - metabolism Cytoplasm - metabolism E-cadherin Gastroenterology. Liver. Pancreas. Abdomen HT-29 HT29 Cells Humans Indomethacin Indomethacin - pharmacology Investigative techniques, diagnostic techniques (general aspects) Medical sciences Membrane Proteins - antagonists & inhibitors Membrane Proteins - metabolism Pathology. Cytology. Biochemistry. Spectrometry. Miscellaneous investigative techniques Signal Transduction Stomach. Duodenum. Small intestine. Colon. Rectum. Anus Tumors Wnt Proteins - metabolism β-catenin
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