Tissue Selectivity in Multiple Endocrine Neoplasia Type 1-Associated Tumorigenesis

The phenotype of the multiple endocrine neoplasia type 1 (MEN1) syndrome cannot be explained solely by the expression pattern of the predisposing gene MEN1 and its encoded protein, menin. This review addresses putative factors determining MEN1-associated tissue-selective tumorigenesis. Menin's...

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Veröffentlicht in:	Cancer research (Chicago, Ill.) Ill.), 2009-08, Vol.69 (16), p.6371-6374
Hauptverfasser:	GRACANIN, Ana, DREIJERINK, Koen M. A, VAN DER LUIJT, Rob B, LIPS, Cornelis J. M, HÖPPENER, Jo W. M
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Antineoplastic agents Biological and medical sciences Cell Transformation, Neoplastic - genetics Endocrinopathies Gene Deletion General aspects. Associated endocrine diseases. Endocrine paraneoplasic syndromes Humans Medical sciences Models, Biological Multiple Endocrine Neoplasia Type 1 - complications Multiple Endocrine Neoplasia Type 1 - genetics Neoplasms - etiology Neoplasms - genetics Organ Specificity - genetics Pharmacology. Drug treatments Proto-Oncogene Proteins - genetics Proto-Oncogene Proteins - physiology Tumors
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