Acute administration of ketamine reverses the inhibition of mitochondrial respiratory chain induced by chronic mild stress

Abstract Modulation and dysfunction of the glutamatergic system seems to be involved in depression. Recently a renewed interest in the glutamatergic system as a treatment option for major depression emerged by the finding that the glutamate N -methyl- d -aspartate (NMDA) antagonist ketamine leads to...

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Veröffentlicht in:	Brain research bulletin 2009-08, Vol.79 (6), p.418-421
Hauptverfasser:	Rezin, Gislaine T, Gonçalves, Cinara L, Daufenbach, Juliana F, Fraga, Daiane B, Santos, Patricia M, Ferreira, Gabriela K, Hermani, Fernanda V, Comim, Clarissa M, Quevedo, João, Streck, Emilio L
Format:	Artikel
Sprache:	eng
Schlagworte:	Adrenal Glands - drug effects Adrenal Glands - pathology Animals Body Weight - drug effects Cerebellum - drug effects Cerebellum - enzymology Cerebral Cortex - drug effects Cerebral Cortex - enzymology Chronic mild stress Depression Eating - drug effects Electron Transport - drug effects Electron Transport - physiology Electron Transport Complex I - metabolism Electron Transport Complex III - metabolism Electron Transport Complex IV - metabolism Excitatory Amino Acid Antagonists - pharmacology Ketamine Ketamine - pharmacology Male Mitochondria Mitochondria - drug effects Mitochondria - enzymology Neurology Organ Size Rats Rats, Wistar Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors Respiratory chain Stress, Psychological - drug therapy Stress, Psychological - enzymology Stress, Psychological - pathology
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