A case of stiff-person syndrome, type 1 diabetes, celiac disease and dermatitis herpetiformis

Abstract Antibodies against glutamic acid decarboxylase (GAD) are involved in the pathophysiology of stiff-person syndrome (SPS) and type 1 diabetes. GAD catalyses the conversion of glutamate to gamma-aminobutyric acid (GABA). GABA acts as a neurotransmitter between neurones, while in pancreatic bet...

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Veröffentlicht in:	Clinical neurology and neurosurgery 2009-05, Vol.111 (4), p.384-386
Hauptverfasser:	O'Sullivan, Eoin P, Behan, Lucy-Ann, King, Tom F.J, Hardiman, Orla, Smith, Diarmuid
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult Anti-GAD antibodies Autoimmune diseases Biological and medical sciences Celiac disease Celiac Disease - diagnosis Celiac Disease - physiopathology Comorbidity Dermatitis herpetiformis Dermatitis Herpetiformis - diagnosis Dermatitis Herpetiformis - physiopathology Diabetes Diabetes Mellitus, Type 1 - diagnosis Diabetes Mellitus, Type 1 - physiopathology Disease Family medical history Glutamic acid decarboxylase Humans Hypothyroidism Male Medical sciences Molecular weight Neurology Neurosurgery Stiff-person syndrome Stiff-Person Syndrome - diagnosis Stiff-Person Syndrome - physiopathology Surgery (general aspects). Transplantations, organ and tissue grafts. Graft diseases Thyroid diseases Thyroid gland Type 1 diabetes
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