Butyrate suppresses hypoxia-inducible factor-1 activity in intestinal epithelial cells under hypoxic conditions

Interaction between the products of intestinal bacteria and the intestinal epithelial cells is a key event in understanding the biological, physiological, and pathological functions of the intestinal epithelium. Here, we examined the effect of butyrate, one of the major intestinal bacterial products...

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Veröffentlicht in:	Shock (Augusta, Ga.) Ga.), 2004-11, Vol.22 (5), p.446-452
Hauptverfasser:	MIKI, Keita, UNNO, Naoki, NAGATA, Toshi, UCHIJIMA, Masato, KONNO, Hiroyuki, KOIDE, Yukio, NAKAMURA, Satoshi
Format:	Artikel
Sprache:	eng
Schlagworte:	Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy Animals Biological and medical sciences Blood. Blood and plasma substitutes. Blood products. Blood cells. Blood typing. Plasmapheresis. Apheresis Bone marrow, stem cells transplantation. Graft versus host reaction Butyrates - pharmacology Caco-2 Cells Cell Line Cell Line, Tumor Cells, Cultured Down-Regulation Electrophysiology Epithelial Cells - drug effects Epithelial Cells - metabolism Fatty Acids - chemistry Humans Hypoxia Hypoxia-Inducible Factor 1, alpha Subunit Intensive care medicine Intestines - drug effects Intestines - metabolism Luciferases - metabolism Medical sciences Mucins - metabolism Muscle Proteins - metabolism Peptides - metabolism Plasmids - metabolism Rats Response Elements Reverse Transcriptase Polymerase Chain Reaction Sodium Oxybate - pharmacology Time Factors Transcription Factors - metabolism Transfection Transfusions. Complications. Transfusion reactions. Cell and gene therapy Trefoil Factor-2 Up-Regulation
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