Oxaloacetate restores the long-term potentiation impaired in rat hippocampus CA1 region by 2-vessel occlusion

Various acute brain pathological conditions are characterized by the presence of elevated glutamate concentrations in the brain interstitial fluids. It has been established that a decrease in the blood glutamate level enhances the brain-to-blood efflux of glutamate, removal of which from the brain m...

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Veröffentlicht in:	European journal of pharmacology 2009-02, Vol.604 (1), p.51-57
Hauptverfasser:	Marosi, Máté, Fuzik, János, Nagy, Dávid, Rákos, Gabriella, Kis, Zsolt, Vécsei, László, Toldi, József, Ruban-Matuzani, Angela, Teichberg, Vivian I., Farkas, Tamás
Format:	Artikel
Sprache:	eng
Schlagworte:	2-vessel occlusion Animals Carotid Arteries - pathology Disease Models, Animal Dose-Response Relationship, Drug Excitatory Postsynaptic Potentials - drug effects Glutamate neurotoxicity Glutamate-scavenging Glutamic Acid - blood Glutamic Acid - metabolism Hippocampus - drug effects Hippocampus - pathology Hippocampus - physiopathology Hypoperfusion Ischemic Attack, Transient - metabolism Ischemic Attack, Transient - pathology Ischemic Attack, Transient - physiopathology Ischemic Attack, Transient - prevention & control Long-Term Potentiation - drug effects LTP Male Oxaloacetate Oxaloacetic Acid - pharmacology Rats Rats, Wistar Synaptic Transmission - drug effects
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