lncRNA MIAT promotes luminal B breast cancer cell proliferation, migration, and invasion in vitro

Long noncoding RNAs (lncRNAs) play a role in the emergence and progression of several human tumors, including luminal B breast cancer (BC). The biological functions and potential mechanisms of lncRNA myocardial infarction-associated transcripts (MIAT) in luminal B BC, on the contrary, are unknown. I...

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Veröffentlicht in:Journal of applied genetics 2024-05, Vol.65 (2), p.309-319
Hauptverfasser: Mi, Jintao, Zhang, Hongsheng, Jiang, Xuemei, Yi, Ying, Cao, Weiwei, Song, Chunjiao, Yuan, Chengliang
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container_issue 2
container_start_page 309
container_title Journal of applied genetics
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creator Mi, Jintao
Zhang, Hongsheng
Jiang, Xuemei
Yi, Ying
Cao, Weiwei
Song, Chunjiao
Yuan, Chengliang
description Long noncoding RNAs (lncRNAs) play a role in the emergence and progression of several human tumors, including luminal B breast cancer (BC). The biological functions and potential mechanisms of lncRNA myocardial infarction-associated transcripts (MIAT) in luminal B BC, on the contrary, are unknown. In this work, we used UALCAN database analysis to find high expression of lncRNA MIAT in luminal BC tissues and also confirmed high levels of lncRNA MIAT expression in luminal B BC tissues and cells. In vitro knockdown of MIAT inhibited the proliferation, migration, and invasion of BT474 cells. In addition, we found that miR-150-5p levels were significantly reduced in luminal B BC specimens and cells, and miR-150-5p levels were significantly increased when MIAT was knocked down. And TIMER database analysis showed that MIAT was positively associated with PDL1. Through bioinformatic tools and in vitro experiments, lncRNA MIAT could function as a competitive endogenous RNA (CeRNA) to further regulate programmed cell death ligand 1 (PDL1) expression by directly sponging miR-150-5p. In conclusion, our data suggest that MIAT, an oncogene, may sponge miR-150-5p to regulate PDL1 expression and affect proliferation, migration, and invasion in luminal B BC in vitro.
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subjects Animal Genetics and Genomics
Apoptosis
bioinformatics
Biomedical and Life Sciences
Breast cancer
breast neoplasms
Breast Neoplasms - genetics
Breast Neoplasms - pathology
Cell death
Cell migration
Cell Movement - genetics
Cell proliferation
Cell Proliferation - genetics
Female
Gene Expression Regulation, Neoplastic
Human Genetics
Human Genetics • Original Paper
Humans
Life Sciences
ligands
Microbial Genetics and Genomics
MicroRNAs - genetics
Myocardial infarction
Myocardial Infarction - genetics
neoplasm cells
Non-coding RNA
oncogenes
Plant Genetics and Genomics
programmed cell death
RNA
RNA, Long Noncoding - genetics
RNA, Long Noncoding - metabolism
title lncRNA MIAT promotes luminal B breast cancer cell proliferation, migration, and invasion in vitro
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