METTL14 Mediates Glut3 m6A methylation to improve osteogenesis under oxidative stress condition

Bone remodeling imbalance contributes to osteoporosis. Though current medications enhance osteoblast involvement in bone formation, the underlying pathways remain unclear. This study was aimed to explore the pathways involved in bone formation by osteoblasts, we investigate the protective role of gl...

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Veröffentlicht in:	Redox report : communications in free radical research 2025-12, Vol.30 (1), p.2435241
Hauptverfasser:	Wang, Ying, Yu, Xueying, Sun, Fenyong, Fu, Yan, Hu, Tingting, Shi, Qiqing, Man, Qiuhong
Format:	Artikel
Sprache:	eng
Schlagworte:	Adenosine - analogs & derivatives Adenosine - metabolism Adenosine - pharmacology Animals Cell Differentiation - drug effects Female Glucose Transporter Type 3 - genetics Glucose Transporter Type 3 - metabolism GLUT3 glycolysis Hydrogen Peroxide - metabolism m6A Methylation - drug effects Methyltransferases - genetics Methyltransferases - metabolism METTL14 Mice Osteoblasts - drug effects Osteoblasts - metabolism Osteogenesis - drug effects Osteoporosis Osteoporosis - drug therapy Osteoporosis - metabolism Oxidative stress Oxidative Stress - drug effects
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