NLRX1 attenuates endoplasmic reticulum stress via STING in cardiac hypertrophy

Endoplasmic reticulum stress-induced cell apoptosis is a pivotal mechanism underlying the progression of cardiac hypertrophy. NLRX1, a member of the NOD-like receptor family, modulates various cellular processes, including STING, NF-κB, MAPK pathways, reactive oxygen species production, essential me...

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Veröffentlicht in:	Biochimica et biophysica acta. Molecular cell research 2024-12, Vol.1871 (8), p.119852, Article 119852
Hauptverfasser:	Mi, Keying, Wang, Xiaoyan, Ma, Chao, Tan, Yinghua, Zhao, Gang, Cao, Xinran, Yuan, Haitao
Format:	Artikel
Sprache:	eng
Schlagworte:	Cardiac hypertrophy Endoplasmic reticulum stress NLRX1 STING
Online-Zugang:	Volltext
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