Tumor necrosis factor alpha induces NOX2-dependent reactive oxygen species production in hypothalamic paraventricular nucleus neurons following angiotensin II infusion

There is evidence that tumor necrosis factor alpha (TNFα) influences autonomic processes coordinated within the hypothalamic paraventricular nucleus (PVN), however, the signaling mechanisms subserving TNFα′s actions in this brain area are unclear. In non-neuronal cell types, TNFα has been shown to p...

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Veröffentlicht in:	Neurochemistry international 2024-10, Vol.179, p.105825, Article 105825
Hauptverfasser:	Woods, Clara, Wang, Gang, Milner, Teresa A., Glass, Michael J.
Format:	Artikel
Sprache:	eng
Schlagworte:	Angiotensin II - metabolism Blood pressure Hypertension Hypertension - metabolism Inflammatory cytokines Male Mice Mice, Inbred C57BL Mice, Knockout NADPH oxidase NADPH Oxidase 2 - metabolism Neurons - metabolism Oxidative stress Paraventricular Hypothalamic Nucleus - cytology Paraventricular Hypothalamic Nucleus - metabolism Reactive Oxygen Species - metabolism Receptors, Tumor Necrosis Factor, Type I - genetics Receptors, Tumor Necrosis Factor, Type I - metabolism Tumor Necrosis Factor-alpha - metabolism
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