Embryonic ethanol exposure induces oxidative stress and inflammation in zebrafish model: A dose-dependent study

Alcohol, or ethanol, is a major contributor to detrimental diseases and comorbidities worldwide. Alcohol use during pregnancy intervenes the developing embryos leading to morphological changes, neurocognitive defects, and behavioral changes known as fetal alcohol spectrum disorder (FASD). Zebrafish...

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Veröffentlicht in:	Toxicology (Amsterdam) 2024-08, Vol.506, p.153876, Article 153876
Hauptverfasser:	Raghul Kannan, Sampath, Latha Laxmi, Indrani Paramasivan, Ahmad, Sheikh F., Tamizhselvi, Ramasamy
Format:	Artikel
Sprache:	eng
Schlagworte:	abnormal development Alcohol Apoptosis body length Danio rerio developmental toxicity dose response ethanol FASD fetal alcohol syndrome gene expression genes glutathione heart rate inflammation lipid peroxidation Malformations nitric oxide oxidative stress pregnancy reactive oxygen species survival rate tail Zebrafish embryos
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