A Rare Case of Extensive Hydrofluoric Acid Burn

Abstract Hydrofluoric acid (HF) is a strongly corrosive, highly toxic, and highly dangerous mineral acid. Burns with over 1% TBSA caused by anhydrous HF can lead to deep tissue damage, hypocalcemia, poisoning, and even death. In recent years, HF has become one of the most common substances causing c...

Ausführliche Beschreibung

Gespeichert in:
Bibliographische Detailangaben
Veröffentlicht in:Journal of burn care & research 2024-09, Vol.45 (5), p.1321-1324
Hauptverfasser: Hu, Gaozhong, Shu, Ziqin, Li, Yuan, Song, Huapei
Format: Artikel
Sprache:eng
Schlagworte:
Online-Zugang:Volltext
Tags: Tag hinzufügen
Keine Tags, Fügen Sie den ersten Tag hinzu!
container_end_page 1324
container_issue 5
container_start_page 1321
container_title Journal of burn care & research
container_volume 45
creator Hu, Gaozhong
Shu, Ziqin
Li, Yuan
Song, Huapei
description Abstract Hydrofluoric acid (HF) is a strongly corrosive, highly toxic, and highly dangerous mineral acid. Burns with over 1% TBSA caused by anhydrous HF can lead to deep tissue damage, hypocalcemia, poisoning, and even death. In recent years, HF has become one of the most common substances causing chemical burns and ranks as the leading cause of death from chemical burns. Herein, we report a rare case with 91% TBSA burns caused by 35% HF. The patient developed complications such as shock, severe hypocalcemia, metabolic acidosis, and respiratory failure. Multidisciplinary team consultation (burns, respiratory medicine, nephrology, infectious disease, and pharmacy) was performed immediately after admission. An individualized diagnosis and treatment plan were developed for the patient. The patient was given intensive care, blood volume monitoring, tracheotomy, fluid resuscitation, continuous blood purification, anti-infective and analgesic treatments, intravenous and percutaneous calcium supplementation, early rehabilitation training, psychological rehabilitation, and other treatments. To prevent the wound from deepening, large-area debridement and skin grafting were performed early after the injury. A large dose of 10% calcium gluconate was injected into the patient in divided doses, and the wound was continuously treated with wet dressings. Multiple surgical debridements, negative pressure wound treatment, biological dressings, and Meek skin grafting were performed. After most of the wounds (approximately 85% TBSA) healed, the patient was discharged from the hospital and continued to undergo dressing changes at a local hospital. The patient was followed up 3 months after discharge. All the wounds healed well, and the patient basically regained functional independence in daily life.
doi_str_mv 10.1093/jbcr/irae104
format Article
fullrecord <record><control><sourceid>proquest_cross</sourceid><recordid>TN_cdi_proquest_miscellaneous_3065271656</recordid><sourceformat>XML</sourceformat><sourcesystem>PC</sourcesystem><oup_id>10.1093/jbcr/irae104</oup_id><sourcerecordid>3065271656</sourcerecordid><originalsourceid>FETCH-LOGICAL-c210t-81c60ab62a954b08538ab2e74b9260312b4c77b9ab141230cb8b8268ed3954883</originalsourceid><addsrcrecordid>eNp9kL1LAzEYh4MotlY3Z8mmg2fffF5urEe1QkEQBbeQ5HJw5drUpCf2v_dKa0en9x2e3zM8CF0TeCBQsPHCujhuovEE-AkaEiGKDLhSp8c__xygi5QWAJxDLs7RgCnFqVB0iMYT_Gaix6VJHocaT382fpWab49n2yqGuu1CbByeuKbCj11cXaKz2rTJXx3uCH08Td_LWTZ_fX4pJ_PMUQKbTBEnwVhJTSG4BSWYMpb6nNuCSmCEWu7y3BbGEk4oA2eVVVQqX7F-oBQbobu9dx3DV-fTRi-b5HzbmpUPXdIMpKA5kUL26P0edTGkFH2t17FZmrjVBPQukd4l0odEPX5zMHd26asj_NekB273QOjW_6t-AXiPbbk</addsrcrecordid><sourcetype>Aggregation Database</sourcetype><iscdi>true</iscdi><recordtype>article</recordtype><pqid>3065271656</pqid></control><display><type>article</type><title>A Rare Case of Extensive Hydrofluoric Acid Burn</title><source>MEDLINE</source><source>Oxford University Press Journals All Titles (1996-Current)</source><creator>Hu, Gaozhong ; Shu, Ziqin ; Li, Yuan ; Song, Huapei</creator><creatorcontrib>Hu, Gaozhong ; Shu, Ziqin ; Li, Yuan ; Song, Huapei</creatorcontrib><description>Abstract Hydrofluoric acid (HF) is a strongly corrosive, highly toxic, and highly dangerous mineral acid. Burns with over 1% TBSA caused by anhydrous HF can lead to deep tissue damage, hypocalcemia, poisoning, and even death. In recent years, HF has become one of the most common substances causing chemical burns and ranks as the leading cause of death from chemical burns. Herein, we report a rare case with 91% TBSA burns caused by 35% HF. The patient developed complications such as shock, severe hypocalcemia, metabolic acidosis, and respiratory failure. Multidisciplinary team consultation (burns, respiratory medicine, nephrology, infectious disease, and pharmacy) was performed immediately after admission. An individualized diagnosis and treatment plan were developed for the patient. The patient was given intensive care, blood volume monitoring, tracheotomy, fluid resuscitation, continuous blood purification, anti-infective and analgesic treatments, intravenous and percutaneous calcium supplementation, early rehabilitation training, psychological rehabilitation, and other treatments. To prevent the wound from deepening, large-area debridement and skin grafting were performed early after the injury. A large dose of 10% calcium gluconate was injected into the patient in divided doses, and the wound was continuously treated with wet dressings. Multiple surgical debridements, negative pressure wound treatment, biological dressings, and Meek skin grafting were performed. After most of the wounds (approximately 85% TBSA) healed, the patient was discharged from the hospital and continued to undergo dressing changes at a local hospital. The patient was followed up 3 months after discharge. All the wounds healed well, and the patient basically regained functional independence in daily life.</description><identifier>ISSN: 1559-047X</identifier><identifier>ISSN: 1559-0488</identifier><identifier>EISSN: 1559-0488</identifier><identifier>DOI: 10.1093/jbcr/irae104</identifier><identifier>PMID: 38842582</identifier><language>eng</language><publisher>US: Oxford University Press</publisher><subject>Adult ; Burns, Chemical - etiology ; Burns, Chemical - therapy ; Calcium Gluconate - therapeutic use ; Debridement ; Humans ; Hydrofluoric Acid ; Male ; Skin Transplantation</subject><ispartof>Journal of burn care &amp; research, 2024-09, Vol.45 (5), p.1321-1324</ispartof><rights>The Author(s) 2024. Published by Oxford University Press on behalf of the American Burn Association. All rights reserved. For commercial re-use, please contact reprints@oup.com for reprints and translation rights for reprints. All other permissions can be obtained through our RightsLink service via the Permissions link on the article page on our site—for further information please contact journals.permissions@oup.com. 2024</rights><rights>The Author(s) 2024. Published by Oxford University Press on behalf of the American Burn Association. All rights reserved. For commercial re-use, please contact reprints@oup.com for reprints and translation rights for reprints. All other permissions can be obtained through our RightsLink service via the Permissions link on the article page on our site—for further information please contact journals.permissions@oup.com.</rights><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed><cites>FETCH-LOGICAL-c210t-81c60ab62a954b08538ab2e74b9260312b4c77b9ab141230cb8b8268ed3954883</cites><orcidid>0000-0001-5157-239X</orcidid></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>315,781,785,1585,27929,27930</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/38842582$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Hu, Gaozhong</creatorcontrib><creatorcontrib>Shu, Ziqin</creatorcontrib><creatorcontrib>Li, Yuan</creatorcontrib><creatorcontrib>Song, Huapei</creatorcontrib><title>A Rare Case of Extensive Hydrofluoric Acid Burn</title><title>Journal of burn care &amp; research</title><addtitle>J Burn Care Res</addtitle><description>Abstract Hydrofluoric acid (HF) is a strongly corrosive, highly toxic, and highly dangerous mineral acid. Burns with over 1% TBSA caused by anhydrous HF can lead to deep tissue damage, hypocalcemia, poisoning, and even death. In recent years, HF has become one of the most common substances causing chemical burns and ranks as the leading cause of death from chemical burns. Herein, we report a rare case with 91% TBSA burns caused by 35% HF. The patient developed complications such as shock, severe hypocalcemia, metabolic acidosis, and respiratory failure. Multidisciplinary team consultation (burns, respiratory medicine, nephrology, infectious disease, and pharmacy) was performed immediately after admission. An individualized diagnosis and treatment plan were developed for the patient. The patient was given intensive care, blood volume monitoring, tracheotomy, fluid resuscitation, continuous blood purification, anti-infective and analgesic treatments, intravenous and percutaneous calcium supplementation, early rehabilitation training, psychological rehabilitation, and other treatments. To prevent the wound from deepening, large-area debridement and skin grafting were performed early after the injury. A large dose of 10% calcium gluconate was injected into the patient in divided doses, and the wound was continuously treated with wet dressings. Multiple surgical debridements, negative pressure wound treatment, biological dressings, and Meek skin grafting were performed. After most of the wounds (approximately 85% TBSA) healed, the patient was discharged from the hospital and continued to undergo dressing changes at a local hospital. The patient was followed up 3 months after discharge. All the wounds healed well, and the patient basically regained functional independence in daily life.</description><subject>Adult</subject><subject>Burns, Chemical - etiology</subject><subject>Burns, Chemical - therapy</subject><subject>Calcium Gluconate - therapeutic use</subject><subject>Debridement</subject><subject>Humans</subject><subject>Hydrofluoric Acid</subject><subject>Male</subject><subject>Skin Transplantation</subject><issn>1559-047X</issn><issn>1559-0488</issn><issn>1559-0488</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2024</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNp9kL1LAzEYh4MotlY3Z8mmg2fffF5urEe1QkEQBbeQ5HJw5drUpCf2v_dKa0en9x2e3zM8CF0TeCBQsPHCujhuovEE-AkaEiGKDLhSp8c__xygi5QWAJxDLs7RgCnFqVB0iMYT_Gaix6VJHocaT382fpWab49n2yqGuu1CbByeuKbCj11cXaKz2rTJXx3uCH08Td_LWTZ_fX4pJ_PMUQKbTBEnwVhJTSG4BSWYMpb6nNuCSmCEWu7y3BbGEk4oA2eVVVQqX7F-oBQbobu9dx3DV-fTRi-b5HzbmpUPXdIMpKA5kUL26P0edTGkFH2t17FZmrjVBPQukd4l0odEPX5zMHd26asj_NekB273QOjW_6t-AXiPbbk</recordid><startdate>20240906</startdate><enddate>20240906</enddate><creator>Hu, Gaozhong</creator><creator>Shu, Ziqin</creator><creator>Li, Yuan</creator><creator>Song, Huapei</creator><general>Oxford University Press</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope><orcidid>https://orcid.org/0000-0001-5157-239X</orcidid></search><sort><creationdate>20240906</creationdate><title>A Rare Case of Extensive Hydrofluoric Acid Burn</title><author>Hu, Gaozhong ; Shu, Ziqin ; Li, Yuan ; Song, Huapei</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c210t-81c60ab62a954b08538ab2e74b9260312b4c77b9ab141230cb8b8268ed3954883</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2024</creationdate><topic>Adult</topic><topic>Burns, Chemical - etiology</topic><topic>Burns, Chemical - therapy</topic><topic>Calcium Gluconate - therapeutic use</topic><topic>Debridement</topic><topic>Humans</topic><topic>Hydrofluoric Acid</topic><topic>Male</topic><topic>Skin Transplantation</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Hu, Gaozhong</creatorcontrib><creatorcontrib>Shu, Ziqin</creatorcontrib><creatorcontrib>Li, Yuan</creatorcontrib><creatorcontrib>Song, Huapei</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Journal of burn care &amp; research</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Hu, Gaozhong</au><au>Shu, Ziqin</au><au>Li, Yuan</au><au>Song, Huapei</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>A Rare Case of Extensive Hydrofluoric Acid Burn</atitle><jtitle>Journal of burn care &amp; research</jtitle><addtitle>J Burn Care Res</addtitle><date>2024-09-06</date><risdate>2024</risdate><volume>45</volume><issue>5</issue><spage>1321</spage><epage>1324</epage><pages>1321-1324</pages><issn>1559-047X</issn><issn>1559-0488</issn><eissn>1559-0488</eissn><abstract>Abstract Hydrofluoric acid (HF) is a strongly corrosive, highly toxic, and highly dangerous mineral acid. Burns with over 1% TBSA caused by anhydrous HF can lead to deep tissue damage, hypocalcemia, poisoning, and even death. In recent years, HF has become one of the most common substances causing chemical burns and ranks as the leading cause of death from chemical burns. Herein, we report a rare case with 91% TBSA burns caused by 35% HF. The patient developed complications such as shock, severe hypocalcemia, metabolic acidosis, and respiratory failure. Multidisciplinary team consultation (burns, respiratory medicine, nephrology, infectious disease, and pharmacy) was performed immediately after admission. An individualized diagnosis and treatment plan were developed for the patient. The patient was given intensive care, blood volume monitoring, tracheotomy, fluid resuscitation, continuous blood purification, anti-infective and analgesic treatments, intravenous and percutaneous calcium supplementation, early rehabilitation training, psychological rehabilitation, and other treatments. To prevent the wound from deepening, large-area debridement and skin grafting were performed early after the injury. A large dose of 10% calcium gluconate was injected into the patient in divided doses, and the wound was continuously treated with wet dressings. Multiple surgical debridements, negative pressure wound treatment, biological dressings, and Meek skin grafting were performed. After most of the wounds (approximately 85% TBSA) healed, the patient was discharged from the hospital and continued to undergo dressing changes at a local hospital. The patient was followed up 3 months after discharge. All the wounds healed well, and the patient basically regained functional independence in daily life.</abstract><cop>US</cop><pub>Oxford University Press</pub><pmid>38842582</pmid><doi>10.1093/jbcr/irae104</doi><tpages>4</tpages><orcidid>https://orcid.org/0000-0001-5157-239X</orcidid></addata></record>
fulltext fulltext
identifier ISSN: 1559-047X
ispartof Journal of burn care & research, 2024-09, Vol.45 (5), p.1321-1324
issn 1559-047X
1559-0488
1559-0488
language eng
recordid cdi_proquest_miscellaneous_3065271656
source MEDLINE; Oxford University Press Journals All Titles (1996-Current)
subjects Adult
Burns, Chemical - etiology
Burns, Chemical - therapy
Calcium Gluconate - therapeutic use
Debridement
Humans
Hydrofluoric Acid
Male
Skin Transplantation
title A Rare Case of Extensive Hydrofluoric Acid Burn
url https://sfx.bib-bvb.de/sfx_tum?ctx_ver=Z39.88-2004&ctx_enc=info:ofi/enc:UTF-8&ctx_tim=2024-12-14T12%3A53%3A19IST&url_ver=Z39.88-2004&url_ctx_fmt=infofi/fmt:kev:mtx:ctx&rfr_id=info:sid/primo.exlibrisgroup.com:primo3-Article-proquest_cross&rft_val_fmt=info:ofi/fmt:kev:mtx:journal&rft.genre=article&rft.atitle=A%20Rare%20Case%20of%20Extensive%20Hydrofluoric%20Acid%20Burn&rft.jtitle=Journal%20of%20burn%20care%20&%20research&rft.au=Hu,%20Gaozhong&rft.date=2024-09-06&rft.volume=45&rft.issue=5&rft.spage=1321&rft.epage=1324&rft.pages=1321-1324&rft.issn=1559-047X&rft.eissn=1559-0488&rft_id=info:doi/10.1093/jbcr/irae104&rft_dat=%3Cproquest_cross%3E3065271656%3C/proquest_cross%3E%3Curl%3E%3C/url%3E&disable_directlink=true&sfx.directlink=off&sfx.report_link=0&rft_id=info:oai/&rft_pqid=3065271656&rft_id=info:pmid/38842582&rft_oup_id=10.1093/jbcr/irae104&rfr_iscdi=true