A Rare Case of Extensive Hydrofluoric Acid Burn
Abstract Hydrofluoric acid (HF) is a strongly corrosive, highly toxic, and highly dangerous mineral acid. Burns with over 1% TBSA caused by anhydrous HF can lead to deep tissue damage, hypocalcemia, poisoning, and even death. In recent years, HF has become one of the most common substances causing c...
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creator | Hu, Gaozhong Shu, Ziqin Li, Yuan Song, Huapei |
description | Abstract
Hydrofluoric acid (HF) is a strongly corrosive, highly toxic, and highly dangerous mineral acid. Burns with over 1% TBSA caused by anhydrous HF can lead to deep tissue damage, hypocalcemia, poisoning, and even death. In recent years, HF has become one of the most common substances causing chemical burns and ranks as the leading cause of death from chemical burns. Herein, we report a rare case with 91% TBSA burns caused by 35% HF. The patient developed complications such as shock, severe hypocalcemia, metabolic acidosis, and respiratory failure. Multidisciplinary team consultation (burns, respiratory medicine, nephrology, infectious disease, and pharmacy) was performed immediately after admission. An individualized diagnosis and treatment plan were developed for the patient. The patient was given intensive care, blood volume monitoring, tracheotomy, fluid resuscitation, continuous blood purification, anti-infective and analgesic treatments, intravenous and percutaneous calcium supplementation, early rehabilitation training, psychological rehabilitation, and other treatments. To prevent the wound from deepening, large-area debridement and skin grafting were performed early after the injury. A large dose of 10% calcium gluconate was injected into the patient in divided doses, and the wound was continuously treated with wet dressings. Multiple surgical debridements, negative pressure wound treatment, biological dressings, and Meek skin grafting were performed. After most of the wounds (approximately 85% TBSA) healed, the patient was discharged from the hospital and continued to undergo dressing changes at a local hospital. The patient was followed up 3 months after discharge. All the wounds healed well, and the patient basically regained functional independence in daily life. |
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Hydrofluoric acid (HF) is a strongly corrosive, highly toxic, and highly dangerous mineral acid. Burns with over 1% TBSA caused by anhydrous HF can lead to deep tissue damage, hypocalcemia, poisoning, and even death. In recent years, HF has become one of the most common substances causing chemical burns and ranks as the leading cause of death from chemical burns. Herein, we report a rare case with 91% TBSA burns caused by 35% HF. The patient developed complications such as shock, severe hypocalcemia, metabolic acidosis, and respiratory failure. Multidisciplinary team consultation (burns, respiratory medicine, nephrology, infectious disease, and pharmacy) was performed immediately after admission. An individualized diagnosis and treatment plan were developed for the patient. The patient was given intensive care, blood volume monitoring, tracheotomy, fluid resuscitation, continuous blood purification, anti-infective and analgesic treatments, intravenous and percutaneous calcium supplementation, early rehabilitation training, psychological rehabilitation, and other treatments. To prevent the wound from deepening, large-area debridement and skin grafting were performed early after the injury. A large dose of 10% calcium gluconate was injected into the patient in divided doses, and the wound was continuously treated with wet dressings. Multiple surgical debridements, negative pressure wound treatment, biological dressings, and Meek skin grafting were performed. After most of the wounds (approximately 85% TBSA) healed, the patient was discharged from the hospital and continued to undergo dressing changes at a local hospital. The patient was followed up 3 months after discharge. All the wounds healed well, and the patient basically regained functional independence in daily life.</description><identifier>ISSN: 1559-047X</identifier><identifier>ISSN: 1559-0488</identifier><identifier>EISSN: 1559-0488</identifier><identifier>DOI: 10.1093/jbcr/irae104</identifier><identifier>PMID: 38842582</identifier><language>eng</language><publisher>US: Oxford University Press</publisher><subject>Adult ; Burns, Chemical - etiology ; Burns, Chemical - therapy ; Calcium Gluconate - therapeutic use ; Debridement ; Humans ; Hydrofluoric Acid ; Male ; Skin Transplantation</subject><ispartof>Journal of burn care & research, 2024-09, Vol.45 (5), p.1321-1324</ispartof><rights>The Author(s) 2024. Published by Oxford University Press on behalf of the American Burn Association. All rights reserved. For commercial re-use, please contact reprints@oup.com for reprints and translation rights for reprints. All other permissions can be obtained through our RightsLink service via the Permissions link on the article page on our site—for further information please contact journals.permissions@oup.com. 2024</rights><rights>The Author(s) 2024. Published by Oxford University Press on behalf of the American Burn Association. All rights reserved. For commercial re-use, please contact reprints@oup.com for reprints and translation rights for reprints. All other permissions can be obtained through our RightsLink service via the Permissions link on the article page on our site—for further information please contact journals.permissions@oup.com.</rights><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed><cites>FETCH-LOGICAL-c210t-81c60ab62a954b08538ab2e74b9260312b4c77b9ab141230cb8b8268ed3954883</cites><orcidid>0000-0001-5157-239X</orcidid></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>315,781,785,1585,27929,27930</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/38842582$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Hu, Gaozhong</creatorcontrib><creatorcontrib>Shu, Ziqin</creatorcontrib><creatorcontrib>Li, Yuan</creatorcontrib><creatorcontrib>Song, Huapei</creatorcontrib><title>A Rare Case of Extensive Hydrofluoric Acid Burn</title><title>Journal of burn care & research</title><addtitle>J Burn Care Res</addtitle><description>Abstract
Hydrofluoric acid (HF) is a strongly corrosive, highly toxic, and highly dangerous mineral acid. Burns with over 1% TBSA caused by anhydrous HF can lead to deep tissue damage, hypocalcemia, poisoning, and even death. In recent years, HF has become one of the most common substances causing chemical burns and ranks as the leading cause of death from chemical burns. Herein, we report a rare case with 91% TBSA burns caused by 35% HF. The patient developed complications such as shock, severe hypocalcemia, metabolic acidosis, and respiratory failure. Multidisciplinary team consultation (burns, respiratory medicine, nephrology, infectious disease, and pharmacy) was performed immediately after admission. An individualized diagnosis and treatment plan were developed for the patient. The patient was given intensive care, blood volume monitoring, tracheotomy, fluid resuscitation, continuous blood purification, anti-infective and analgesic treatments, intravenous and percutaneous calcium supplementation, early rehabilitation training, psychological rehabilitation, and other treatments. To prevent the wound from deepening, large-area debridement and skin grafting were performed early after the injury. A large dose of 10% calcium gluconate was injected into the patient in divided doses, and the wound was continuously treated with wet dressings. Multiple surgical debridements, negative pressure wound treatment, biological dressings, and Meek skin grafting were performed. After most of the wounds (approximately 85% TBSA) healed, the patient was discharged from the hospital and continued to undergo dressing changes at a local hospital. The patient was followed up 3 months after discharge. All the wounds healed well, and the patient basically regained functional independence in daily life.</description><subject>Adult</subject><subject>Burns, Chemical - etiology</subject><subject>Burns, Chemical - therapy</subject><subject>Calcium Gluconate - therapeutic use</subject><subject>Debridement</subject><subject>Humans</subject><subject>Hydrofluoric Acid</subject><subject>Male</subject><subject>Skin Transplantation</subject><issn>1559-047X</issn><issn>1559-0488</issn><issn>1559-0488</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2024</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNp9kL1LAzEYh4MotlY3Z8mmg2fffF5urEe1QkEQBbeQ5HJw5drUpCf2v_dKa0en9x2e3zM8CF0TeCBQsPHCujhuovEE-AkaEiGKDLhSp8c__xygi5QWAJxDLs7RgCnFqVB0iMYT_Gaix6VJHocaT382fpWab49n2yqGuu1CbByeuKbCj11cXaKz2rTJXx3uCH08Td_LWTZ_fX4pJ_PMUQKbTBEnwVhJTSG4BSWYMpb6nNuCSmCEWu7y3BbGEk4oA2eVVVQqX7F-oBQbobu9dx3DV-fTRi-b5HzbmpUPXdIMpKA5kUL26P0edTGkFH2t17FZmrjVBPQukd4l0odEPX5zMHd26asj_NekB273QOjW_6t-AXiPbbk</recordid><startdate>20240906</startdate><enddate>20240906</enddate><creator>Hu, Gaozhong</creator><creator>Shu, Ziqin</creator><creator>Li, Yuan</creator><creator>Song, Huapei</creator><general>Oxford University Press</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope><orcidid>https://orcid.org/0000-0001-5157-239X</orcidid></search><sort><creationdate>20240906</creationdate><title>A Rare Case of Extensive Hydrofluoric Acid Burn</title><author>Hu, Gaozhong ; Shu, Ziqin ; Li, Yuan ; Song, Huapei</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c210t-81c60ab62a954b08538ab2e74b9260312b4c77b9ab141230cb8b8268ed3954883</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2024</creationdate><topic>Adult</topic><topic>Burns, Chemical - etiology</topic><topic>Burns, Chemical - therapy</topic><topic>Calcium Gluconate - therapeutic use</topic><topic>Debridement</topic><topic>Humans</topic><topic>Hydrofluoric Acid</topic><topic>Male</topic><topic>Skin Transplantation</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Hu, Gaozhong</creatorcontrib><creatorcontrib>Shu, Ziqin</creatorcontrib><creatorcontrib>Li, Yuan</creatorcontrib><creatorcontrib>Song, Huapei</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Journal of burn care & research</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Hu, Gaozhong</au><au>Shu, Ziqin</au><au>Li, Yuan</au><au>Song, Huapei</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>A Rare Case of Extensive Hydrofluoric Acid Burn</atitle><jtitle>Journal of burn care & research</jtitle><addtitle>J Burn Care Res</addtitle><date>2024-09-06</date><risdate>2024</risdate><volume>45</volume><issue>5</issue><spage>1321</spage><epage>1324</epage><pages>1321-1324</pages><issn>1559-047X</issn><issn>1559-0488</issn><eissn>1559-0488</eissn><abstract>Abstract
Hydrofluoric acid (HF) is a strongly corrosive, highly toxic, and highly dangerous mineral acid. Burns with over 1% TBSA caused by anhydrous HF can lead to deep tissue damage, hypocalcemia, poisoning, and even death. In recent years, HF has become one of the most common substances causing chemical burns and ranks as the leading cause of death from chemical burns. Herein, we report a rare case with 91% TBSA burns caused by 35% HF. The patient developed complications such as shock, severe hypocalcemia, metabolic acidosis, and respiratory failure. Multidisciplinary team consultation (burns, respiratory medicine, nephrology, infectious disease, and pharmacy) was performed immediately after admission. An individualized diagnosis and treatment plan were developed for the patient. The patient was given intensive care, blood volume monitoring, tracheotomy, fluid resuscitation, continuous blood purification, anti-infective and analgesic treatments, intravenous and percutaneous calcium supplementation, early rehabilitation training, psychological rehabilitation, and other treatments. To prevent the wound from deepening, large-area debridement and skin grafting were performed early after the injury. A large dose of 10% calcium gluconate was injected into the patient in divided doses, and the wound was continuously treated with wet dressings. Multiple surgical debridements, negative pressure wound treatment, biological dressings, and Meek skin grafting were performed. After most of the wounds (approximately 85% TBSA) healed, the patient was discharged from the hospital and continued to undergo dressing changes at a local hospital. The patient was followed up 3 months after discharge. All the wounds healed well, and the patient basically regained functional independence in daily life.</abstract><cop>US</cop><pub>Oxford University Press</pub><pmid>38842582</pmid><doi>10.1093/jbcr/irae104</doi><tpages>4</tpages><orcidid>https://orcid.org/0000-0001-5157-239X</orcidid></addata></record> |
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source | MEDLINE; Oxford University Press Journals All Titles (1996-Current) |
subjects | Adult Burns, Chemical - etiology Burns, Chemical - therapy Calcium Gluconate - therapeutic use Debridement Humans Hydrofluoric Acid Male Skin Transplantation |
title | A Rare Case of Extensive Hydrofluoric Acid Burn |
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