Golm1 facilitates the CaO2-DOPC-DSPE200-PEI -CsPbBr3 QDs -induced apoptotic death of hepatocytes through the stimulation of mitochondrial autophagy and mitochondrial reactive oxygen species production through interactions with P53/Beclin-1/Bcl-2

Mitophagy is a distinct physiological process that can have beneficial or deleterious effects in particular tissues. Prior research suggests that mitophagic activity can be triggered by CaO2-PM-CsPbBr3 QDs, yet the specific role that mitophagy plays in hepatic injury induced by CaO2-PM-CsPbBr3 QDs h...

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Veröffentlicht in:	Chemico-biological interactions 2024-08, Vol.398, p.111076, Article 111076
Hauptverfasser:	Zhang, Zhiqiang, Wang, Qinglong, Zhang, Haibo, Wang, Shengchao, Ma, Xia, Wang, Hui
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Apoptosis Autophagy Beclin-1 - metabolism CaO2-PM-CsPbBr3 QDs Golm1 Hepatocytes - cytology Hepatocytes - metabolism Humans Male Membrane Proteins - genetics Membrane Proteins - metabolism Mice Mice, Inbred C57BL Mitochondria - metabolism Mitochondrial autophagy Mitophagy mtROS Proto-Oncogene Proteins c-bcl-2 - metabolism Quantum Dots - chemistry Reactive Oxygen Species - metabolism Tumor Suppressor Protein p53 - metabolism
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