The role of alcohol in work-related fatal accidents in Australia 1982–1984
This paper describes the role of detectable blood alcohol in fatal work injuries. An attempt was made to identify all work-related fatalities that occurred throughout Australia in the period 1982–1984. A research team examined coroners' records and classified 1737 fatal injury cases as being wo...
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Veröffentlicht in: | Occupational medicine (Oxford) 1993, Vol.43 (1), p.13-17 |
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description | This paper describes the role of detectable blood alcohol in fatal work injuries. An attempt was made to identify all work-related fatalities that occurred throughout Australia in the period 1982–1984. A research team examined coroners' records and classified 1737 fatal injury cases as being work-related according to study definitions. The following were also extracted from data in coroners' records: (i) whether or not the description of the fatal event indicated that inebriation was likely to have been a factor; (ii) whether or not there was documentation of blood alcohol concentration (BAC), and if so, what it was. The likelihood of inebriation was assessed without knowledge of the victim's BAC. In 1030 (59 per cent) of the 1737 fatal work injury cases, a BAC determination was documented. Zero levels were detected in 867 fatalities (84 per cent), and 163 cases (16 per cent) had non-zero BAC. In the latter group the median BAC was 104 mg%. Sixty-five per cent of measurable BAC cases had BAC greater than 50 mg%. Fatality risk in the non-zero BAC group relative to that of the zero BAC group was elevated for the following factors: marital status-single (risk ratio (RR)=1.7, 95 per cent confidence interval (Cl) 1.1–2.8) or separated/divorced (RR=2.4, Cl 1.5–3.8); occupation as manager, executive or administrator (RR=2.5, Cl 1.5–5.8); and commuting (RR=1.6, Cl 1.2–2.0). In fatal vehicle accidents, BAC≥50mg% was measured significantly more frequently (RR=1.6, Cl 1.2–2.0) and BAC |
doi_str_mv | 10.1093/occmed/43.1.13 |
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An attempt was made to identify all work-related fatalities that occurred throughout Australia in the period 1982–1984. A research team examined coroners' records and classified 1737 fatal injury cases as being work-related according to study definitions. The following were also extracted from data in coroners' records: (i) whether or not the description of the fatal event indicated that inebriation was likely to have been a factor; (ii) whether or not there was documentation of blood alcohol concentration (BAC), and if so, what it was. The likelihood of inebriation was assessed without knowledge of the victim's BAC. In 1030 (59 per cent) of the 1737 fatal work injury cases, a BAC determination was documented. Zero levels were detected in 867 fatalities (84 per cent), and 163 cases (16 per cent) had non-zero BAC. In the latter group the median BAC was 104 mg%. Sixty-five per cent of measurable BAC cases had BAC greater than 50 mg%. Fatality risk in the non-zero BAC group relative to that of the zero BAC group was elevated for the following factors: marital status-single (risk ratio (RR)=1.7, 95 per cent confidence interval (Cl) 1.1–2.8) or separated/divorced (RR=2.4, Cl 1.5–3.8); occupation as manager, executive or administrator (RR=2.5, Cl 1.5–5.8); and commuting (RR=1.6, Cl 1.2–2.0). In fatal vehicle accidents, BAC≥50mg% was measured significantly more frequently (RR=1.6, Cl 1.2–2.0) and BAC<50 mg% less frequently (RR=0.5, Cl 0.2–0.9) than BAC=0, while non-vehicular workplace accidents were less likely to have involved a high BAC</description><identifier>ISSN: 0962-7480</identifier><identifier>EISSN: 1471-8405</identifier><identifier>DOI: 10.1093/occmed/43.1.13</identifier><identifier>PMID: 8422440</identifier><language>eng</language><publisher>Oxford: Oxford University Press</publisher><subject>Accidents, Occupational - mortality ; Alcoholism and acute alcohol poisoning ; Australia ; Biological and medical sciences ; Ethanol - blood ; Humans ; Medical sciences ; Occupational Diseases - blood ; Occupational Diseases - mortality ; Toxicology ; Work ; Wounds and Injuries - blood ; Wounds and Injuries - mortality</subject><ispartof>Occupational medicine (Oxford), 1993, Vol.43 (1), p.13-17</ispartof><rights>1993 INIST-CNRS</rights><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c450t-e46475eec6aa09bb5d244eafd5988a06b6d010c531ad10e426ac485cc543261e3</citedby></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>314,780,784,4024,27923,27924,27925</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=4599032$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/8422440$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Hollo, Claire D.</creatorcontrib><creatorcontrib>Leigh, James</creatorcontrib><creatorcontrib>Nurminen, Markku</creatorcontrib><title>The role of alcohol in work-related fatal accidents in Australia 1982–1984</title><title>Occupational medicine (Oxford)</title><addtitle>Occup Med (Lond)</addtitle><description>This paper describes the role of detectable blood alcohol in fatal work injuries. An attempt was made to identify all work-related fatalities that occurred throughout Australia in the period 1982–1984. A research team examined coroners' records and classified 1737 fatal injury cases as being work-related according to study definitions. The following were also extracted from data in coroners' records: (i) whether or not the description of the fatal event indicated that inebriation was likely to have been a factor; (ii) whether or not there was documentation of blood alcohol concentration (BAC), and if so, what it was. The likelihood of inebriation was assessed without knowledge of the victim's BAC. In 1030 (59 per cent) of the 1737 fatal work injury cases, a BAC determination was documented. Zero levels were detected in 867 fatalities (84 per cent), and 163 cases (16 per cent) had non-zero BAC. In the latter group the median BAC was 104 mg%. Sixty-five per cent of measurable BAC cases had BAC greater than 50 mg%. Fatality risk in the non-zero BAC group relative to that of the zero BAC group was elevated for the following factors: marital status-single (risk ratio (RR)=1.7, 95 per cent confidence interval (Cl) 1.1–2.8) or separated/divorced (RR=2.4, Cl 1.5–3.8); occupation as manager, executive or administrator (RR=2.5, Cl 1.5–5.8); and commuting (RR=1.6, Cl 1.2–2.0). In fatal vehicle accidents, BAC≥50mg% was measured significantly more frequently (RR=1.6, Cl 1.2–2.0) and BAC<50 mg% less frequently (RR=0.5, Cl 0.2–0.9) than BAC=0, while non-vehicular workplace accidents were less likely to have involved a high BAC</description><subject>Accidents, Occupational - mortality</subject><subject>Alcoholism and acute alcohol poisoning</subject><subject>Australia</subject><subject>Biological and medical sciences</subject><subject>Ethanol - blood</subject><subject>Humans</subject><subject>Medical sciences</subject><subject>Occupational Diseases - blood</subject><subject>Occupational Diseases - mortality</subject><subject>Toxicology</subject><subject>Work</subject><subject>Wounds and Injuries - blood</subject><subject>Wounds and Injuries - mortality</subject><issn>0962-7480</issn><issn>1471-8405</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1993</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNqNkUFvEzEQhS1EVULhyg1pD4jbpmN7bK-PpYIGGolLkRAXa-KdVZc62WJvVLjxH_oP-SVslNArPT1p3jdPo3lCvJIwl-D16RDjmttT1HM5l_qJmEl0sm4QzFMxA29V7bCBZ-J5Kd8BpMVGHYvjBpVChJlYXl1zlYfE1dBVlOJwPaSq31R3Q76pMycaua06GilVFGPf8mYsO_9sW8ZMqadK-kb9-X0_Cb4QRx2lwi8PeiK-fHh_db6ol58vPp6fLeuIBsaa0aIzzNESgV-tTDvdwtS1xjcNgV3ZFiREoyW1EhiVpYiNidGgVlayPhFv97m3efix5TKGdV8ip0QbHrYlKO9ROYBHgNZr49R_welvAEq7CZzvwZiHUjJ34Tb3a8q_goSwKyTsCwmogwxSTwuvD8nb1W7-Dz80MPlvDj6VSKnLtIl9ecDQeA96d2C9x_oy8s8Hm_JNsE47ExZfv4VPXl26y3c2LPRfWMKhhA</recordid><startdate>1993</startdate><enddate>1993</enddate><creator>Hollo, Claire D.</creator><creator>Leigh, James</creator><creator>Nurminen, Markku</creator><general>Oxford University Press</general><scope>BSCLL</scope><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7T2</scope><scope>7U2</scope><scope>C1K</scope><scope>7TB</scope><scope>8FD</scope><scope>FR3</scope><scope>KR7</scope></search><sort><creationdate>1993</creationdate><title>The role of alcohol in work-related fatal accidents in Australia 1982–1984</title><author>Hollo, Claire D. ; Leigh, James ; Nurminen, Markku</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c450t-e46475eec6aa09bb5d244eafd5988a06b6d010c531ad10e426ac485cc543261e3</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1993</creationdate><topic>Accidents, Occupational - mortality</topic><topic>Alcoholism and acute alcohol poisoning</topic><topic>Australia</topic><topic>Biological and medical sciences</topic><topic>Ethanol - blood</topic><topic>Humans</topic><topic>Medical sciences</topic><topic>Occupational Diseases - blood</topic><topic>Occupational Diseases - mortality</topic><topic>Toxicology</topic><topic>Work</topic><topic>Wounds and Injuries - blood</topic><topic>Wounds and Injuries - mortality</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Hollo, Claire D.</creatorcontrib><creatorcontrib>Leigh, James</creatorcontrib><creatorcontrib>Nurminen, Markku</creatorcontrib><collection>Istex</collection><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>Health and Safety Science Abstracts (Full archive)</collection><collection>Safety Science and Risk</collection><collection>Environmental Sciences and Pollution Management</collection><collection>Mechanical & Transportation Engineering Abstracts</collection><collection>Technology Research Database</collection><collection>Engineering Research Database</collection><collection>Civil Engineering Abstracts</collection><jtitle>Occupational medicine (Oxford)</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Hollo, Claire D.</au><au>Leigh, James</au><au>Nurminen, Markku</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>The role of alcohol in work-related fatal accidents in Australia 1982–1984</atitle><jtitle>Occupational medicine (Oxford)</jtitle><addtitle>Occup Med (Lond)</addtitle><date>1993</date><risdate>1993</risdate><volume>43</volume><issue>1</issue><spage>13</spage><epage>17</epage><pages>13-17</pages><issn>0962-7480</issn><eissn>1471-8405</eissn><abstract>This paper describes the role of detectable blood alcohol in fatal work injuries. An attempt was made to identify all work-related fatalities that occurred throughout Australia in the period 1982–1984. A research team examined coroners' records and classified 1737 fatal injury cases as being work-related according to study definitions. The following were also extracted from data in coroners' records: (i) whether or not the description of the fatal event indicated that inebriation was likely to have been a factor; (ii) whether or not there was documentation of blood alcohol concentration (BAC), and if so, what it was. The likelihood of inebriation was assessed without knowledge of the victim's BAC. In 1030 (59 per cent) of the 1737 fatal work injury cases, a BAC determination was documented. Zero levels were detected in 867 fatalities (84 per cent), and 163 cases (16 per cent) had non-zero BAC. In the latter group the median BAC was 104 mg%. Sixty-five per cent of measurable BAC cases had BAC greater than 50 mg%. Fatality risk in the non-zero BAC group relative to that of the zero BAC group was elevated for the following factors: marital status-single (risk ratio (RR)=1.7, 95 per cent confidence interval (Cl) 1.1–2.8) or separated/divorced (RR=2.4, Cl 1.5–3.8); occupation as manager, executive or administrator (RR=2.5, Cl 1.5–5.8); and commuting (RR=1.6, Cl 1.2–2.0). In fatal vehicle accidents, BAC≥50mg% was measured significantly more frequently (RR=1.6, Cl 1.2–2.0) and BAC<50 mg% less frequently (RR=0.5, Cl 0.2–0.9) than BAC=0, while non-vehicular workplace accidents were less likely to have involved a high BAC</abstract><cop>Oxford</cop><pub>Oxford University Press</pub><pmid>8422440</pmid><doi>10.1093/occmed/43.1.13</doi><tpages>5</tpages></addata></record> |
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subjects | Accidents, Occupational - mortality Alcoholism and acute alcohol poisoning Australia Biological and medical sciences Ethanol - blood Humans Medical sciences Occupational Diseases - blood Occupational Diseases - mortality Toxicology Work Wounds and Injuries - blood Wounds and Injuries - mortality |
title | The role of alcohol in work-related fatal accidents in Australia 1982–1984 |
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