MyD88 exacerbates inflammation‐induced bone loss by modulating dynamic equilibrium between Th17/Treg cells and subgingival microbiota dysbiosis

Background This study aimed to investigate the contribution of myeloid differentiation primary‐response gene 88 (MyD88) on the differentiation of T helper type 17 (Th17) and regulatory T (Treg) cells and the emerging subgingival microbiota dysbiosis in Porphyromonas gingivalis‐induced experimental p...

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Veröffentlicht in:	Journal of periodontology (1970) 2024-08, Vol.95 (8), p.764-777
Hauptverfasser:	Hsiao, Po‐Yan, Huang, Ren‐Yeong, Huang, Lin‐Wei, Chu, Ching‐Liang, Dyke, Thomas Van, Mau, Lian‐Ping, Cheng, Chia‐Dan, Sung, Cheng‐En, Weng, Pei‐Wei, Wu, Yu‐Chiao, Shieh, Yi‐Shing, Cheng, Wan‐Chien
Format:	Artikel
Sprache:	eng
Schlagworte:	Alveolar Bone Loss - immunology Alveolar Bone Loss - microbiology Animals Cell Differentiation dysbiosis Dysbiosis - immunology Gingiva - immunology Gingiva - microbiology Inflammation - immunology Male Mice Mice, Inbred C57BL Mice, Knockout Microbiota Myeloid Differentiation Factor 88 Osteoprotegerin - analysis periodontitis Periodontitis - immunology Periodontitis - microbiology Porphyromonas gingivalis RANK Ligand regulatory T lymphocytes T helper 17 cells T-Lymphocytes, Regulatory - immunology Th17 Cells - immunology X-Ray Microtomography - methods
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