The IL-31/TRPV1 pathway mediates allergic asthma exacerbated by DINP dermal exposure in OVA-sensitized Balb/c mice

The potential role of dermal exposure diisononyl phthalate (DINP) as an adjuvant in allergic inflammation and asthma has been suggested. However, the current findings do not provide enough evidence to support this claim. The purpose of this investigation was to examine the impact and mechanisms of a...

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Veröffentlicht in:	The Science of the total environment 2024-02, Vol.912, p.169613-169613, Article 169613
Hauptverfasser:	Peng, Qi, Wu, Yang, Li, Yan, Lu, Chan, Yao, Runming, Hu, Siyuan, Ma, Ning, Chen, Shaohui, Yang, Xu, Ma, Ping
Format:	Artikel
Sprache:	eng
Schlagworte:	adjuvants Allergic asthma antagonists asthma biomarkers compliance Dermal exposure Diisononyl phthalate (DINP) environment histopathology immunoglobulins inflammation interleukin-13 Interleukin-31 (IL-31) interleukin-4 interleukin-5 interleukin-6 lungs phthalates Transient receptor potential vanilloid 1 (TRPV1) transient receptor potential vanilloid channels
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