TNFRSF19 promotes endoplasmic reticulum stress-induced paraptosis via the activation of the MAPK pathway in triple-negative breast cancer cells

TNFRSF19 is a member of the tumor necrosis factor receptor superfamily, and its function exhibits variability among different types of cancers. The influence of TNFRSF19 on triple-negative breast cancer (TNBC) has yet to be definitively established. In this study, bioinformatics analyses revealed th...

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Veröffentlicht in:	Cancer gene therapy 2024-02, Vol.31 (2), p.217-227
Hauptverfasser:	Liu, Shiyang, Tian, Yao, Liu, Chenguang, Gui, Zhengwei, Yu, Tianyao, Zhang, Lin
Format:	Artikel
Sprache:	eng
Schlagworte:	14/19 14/28 14/35 38/1 38/77 42 42/109 631/67/1347 631/67/68 64/60 96/106 96/31 Bioinformatics Biomedical and Life Sciences Biomedicine Breast cancer Caspase Cell death Cell Line, Tumor Cell proliferation Cell Proliferation - genetics Endoplasmic reticulum Endoplasmic Reticulum Stress Gene Expression Gene Therapy Humans In vitro Lymph nodes MAP kinase Medical prognosis Metastases mRNA Paraptosis Protein expression Receptors, Tumor Necrosis Factor - metabolism Therapeutic targets TNF inhibitors Triple Negative Breast Neoplasms - metabolism Tumor suppressor genes
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