Sustained Release of Nitric Oxide‐Mediated Angiogenesis and Nerve Repair by Mussel‐Inspired Adaptable Microreservoirs for Brain Traumatic Injury Therapy

Traumatic brain injury (TBI) triggers inflammatory response and glial scarring, thus substantially hindering brain tissue repair. This process is exacerbated by the accumulation of activated immunocytes at the injury site, which contributes to scar formation and impedes tissue repair. In this study,...

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Veröffentlicht in:	Advanced healthcare materials 2024-10, Vol.13 (25), p.e2302315-n/a
Hauptverfasser:	Liu, Hsiu‐Ching, Huang, Chu‐Han, Chiang, Min‐Ren, Hsu, Ru‐Siou, Chou, Tsu‐Chin, Lu, Tsai‐Te, Lee, I‐Chi, Liao, Lun‐De, Chiou, Shih‐Hwa, Lin, Zhong‐Hong, Hu, Shang‐Hsiu
Format:	Artikel
Sprache:	eng
Schlagworte:	Angiogenesis Animals Bioaccumulation Biocompatibility Bivalvia - chemistry Brain Brain Injuries, Traumatic - drug therapy Brain Injuries, Traumatic - metabolism Brain Injuries, Traumatic - pathology Brain Injuries, Traumatic - therapy Cell adhesion Controlled release Delayed-Action Preparations - chemistry Delayed-Action Preparations - pharmacokinetics Delayed-Action Preparations - pharmacology Head injuries Human Umbilical Vein Endothelial Cells - metabolism Humans Indoles - chemistry Indoles - pharmacology Inflammation Inflammatory response Male Mice Mice, Inbred C57BL Microfluidic devices Microfluidics Neovascularization, Physiologic - drug effects Nerve Regeneration - drug effects nerve repair Neurogenesis Nitric oxide Nitric Oxide - metabolism nitrogen oxide particle scaffold polydopamine Polymers - chemistry Polymers - pharmacology Reactive oxygen species Reactive Oxygen Species - metabolism Repair Structural integrity Sustained release Synergistic effect Therapeutic applications Traumatic brain injury
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