Geniposide attenuates early brain injury by inhibiting oxidative stress and neurocyte apoptosis after subarachnoid hemorrhage in rats

Background Oxidative stress and neurocyte apoptosis are crucial pathophysiological process in early brain injury (EBI) after subarachnoid hemorrhage (SAH). Geniposide (GNP) has been reported to exert neuroprotective effects by reducing oxidative injury and neurocyte apoptosis. However, the effect of...

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Veröffentlicht in:	Molecular biology reports 2022-07, Vol.49 (7), p.6303-6311
Hauptverfasser:	Xiao, Xiaolan, Sun, Shuangxi, Li, Yingbin, Cen, Xuecheng, Wu, Shibiao, Lu, Aili, Cai, Jun, Zhao, Junjie, Li, Shaoxue
Format:	Artikel
Sprache:	eng
Schlagworte:	Animal Anatomy Animal Biochemistry Animal models Animals Apoptosis Biomedical and Life Sciences Blood-brain barrier Brain - metabolism Brain Edema - drug therapy Brain Edema - metabolism Brain Injuries Brain injury Edema Extravasation Glutathione transferase Glutathione Transferase - metabolism Histology Iridoids Life Sciences Male Membrane permeability Morphology Neuroprotection Neuroprotective Agents - metabolism Neuroprotective Agents - pharmacology NF-E2-Related Factor 2 - metabolism Original Article Oxidative Stress Permeability Quinone oxidoreductase Rats Rats, Sprague-Dawley Rats, Wistar Subarachnoid hemorrhage Subarachnoid Hemorrhage - drug therapy Subarachnoid Hemorrhage - metabolism Superoxide dismutase Temporal lobe Traumatic brain injury
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