Aquaporin 4 in Traumatic Brain Injury: From Molecular Pathways to Therapeutic Target

Traumatic brain injury (TBI) is known as an acute degenerative pathology of the central nervous system, and has been shown to increase brain aquaporin 4 (AQP4) expression. Various molecular mechanisms affect AQP4 expression, including neuronal high mobility group box 1, forkhead box O3a, vascular en...

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Veröffentlicht in:	Neurochemical research 2022-04, Vol.47 (4), p.860-871
Hauptverfasser:	Dadgostar, Ehsan, Rahimi, Shiva, Nikmanzar, Shahin, Nazemi, Sina, Naderi Taheri, Mojtaba, Alibolandi, Zahra, Aschner, Michael, Mirzaei, Hamed, Tamtaji, Omid Reza
Format:	Artikel
Sprache:	eng
Schlagworte:	Adenosine Angiotensin Angiotensin II Animals Aquaporin 4 Aquaporin 4 - metabolism Aquaporins Astaxanthin Biochemistry Biomedical and Life Sciences Biomedicine Brain Brain Edema - metabolism Brain Injuries, Traumatic - drug therapy Brain Injuries, Traumatic - metabolism Cell Biology Central nervous system Dizocilpine Edema Epigallocatechin gallate Erythropoietin Estrogens Etiracetam Forkhead protein Gene silencing Glutamic acid receptors Growth factors Head injuries Hypoxia Hypoxia-inducible factor 1 Hypoxia-inducible factor 1a Inactivation Inhibitors Magnesium Molecular modelling N-Methyl-D-aspartic acid receptors Nerve growth factor Neurochemistry Neurology Neurosciences NF-κB protein Poloxamers Progesterone Rats Rats, Sprague-Dawley Receptors Resveratrol Review Tacrolimus Traumatic brain injury Vascular endothelial growth factor Vascular Endothelial Growth Factor A - metabolism
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