Correction of amygdalar dysfunction in a rat model of fragile X syndrome

Fragile X syndrome (FXS), a commonly inherited form of autism and intellectual disability, is associated with emotional symptoms that implicate dysfunction of the amygdala. However, current understanding of the pathogenesis of the disease is based primarily on studies in the hippocampus and neocorte...

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Veröffentlicht in:	Cell reports (Cambridge) 2021-10, Vol.37 (2), p.109805-109805, Article 109805
Hauptverfasser:	Fernandes, Giselle, Mishra, Pradeep K., Nawaz, Mohammad Sarfaraz, Donlin-Asp, Paul G., Rahman, Mohammed Mostafizur, Hazra, Anupam, Kedia, Sonal, Kayenaat, Aiman, Songara, Dheeraj, Wyllie, David J.A., Schuman, Erin M., Kind, Peter C., Chattarji, Sumantra
Format:	Artikel
Sprache:	eng
Schlagworte:	amygdala Animals autism spectrum disorders Basolateral Nuclear Complex - metabolism Basolateral Nuclear Complex - physiopathology Behavior, Animal Disease Models, Animal Fear fear learning Fragile X Mental Retardation Protein - genetics Fragile X Mental Retardation Protein - metabolism Fragile X Syndrome Fragile X Syndrome - genetics Fragile X Syndrome - metabolism Fragile X Syndrome - physiopathology Fragile X Syndrome - psychology long-term potentiation Male Mental Recall mGluR5 Neuronal Plasticity presynaptic Rats Rats, Sprague-Dawley Rats, Transgenic Receptor, Metabotropic Glutamate 5 - metabolism synaptic plasticity Synaptic Transmission
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