Glutamate, Glutamine, GABA and Oxidative Products in the Pons Following Cortical Injury and Their Role in Motor Functional Recovery

Brain injury leads to an excitatory phase followed by an inhibitory phase in the brain. The clinical sequelae caused by cerebral injury seem to be a response to remote functional inhibition of cerebral nuclei located far from the motor cortex but anatomically related to the injury site. It appears t...

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Veröffentlicht in:	Neurochemical research 2021-12, Vol.46 (12), p.3179-3189
Hauptverfasser:	Ramos-Languren, Laura E., Avila-Luna, Alberto, García-Díaz, Gabriela, Rodríguez-Labrada, Roberto, Vázquez-Mojena, Yaimee, Parra-Cid, Carmen, Montes, Sergio, Bueno-Nava, Antonio, González-Piña, Rigoberto
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Apoptosis Biochemistry Biomedical and Life Sciences Biomedicine Brain Brain Injuries - physiopathology Brain injury Brain research Cell Biology Cerebrospinal fluid Complications Cortex (motor) Fluorescence Free radicals gamma-Aminobutyric Acid - metabolism Glutamic Acid - metabolism Glutamine Glutamine - metabolism Head injuries Hypotheses Iron chlorides Lipid Peroxidation Lipids Male Motor activity Motor Cortex - physiology Motor Disorders - physiopathology Motor task performance Neurochemistry Neurology Neurosciences Original Paper Oxidative Stress Peroxidation Pons Pons - metabolism Rats Rats, Wistar Recovery Recovery of Function Sensorimotor system Stroke Traumatic brain injury γ-Aminobutyric acid
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