Ginsenoside F2 induces cellular toxicity to glioblastoma through the impairment of mitochondrial function

Glioblastoma (GBM) is the most aggressive tumor residing within the central nervous system, with extremely poor prognosis. Although the cytotoxic effects of ginsenoside F2 (GF2) on GBM were previously suggested, the precise anti-GBM mechanism of GF2 remains unclear. The aim of this study was to expl...

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Veröffentlicht in:	Phytomedicine (Stuttgart) 2021-03, Vol.83, p.153483-153483, Article 153483
Hauptverfasser:	Kim, Tae-Jun, Kim, Hyeon Ji, Kang, Mingyu, Cho, Jin-Hwa, Kim, Yu Gyung, Lee, Sang Min, Byun, Jin-Seok, Kim, Do-Yeon
Format:	Artikel
Sprache:	eng
Schlagworte:	Antineoplastic Agents, Phytogenic - pharmacology Caspase 3 - metabolism Cell Death - drug effects Cell Line, Tumor Gene Expression Regulation, Neoplastic - drug effects Ginsenoside F2 Ginsenosides - pharmacology Glioblastoma Glioblastoma - drug therapy Glioblastoma - metabolism Glioblastoma - pathology Glutaredoxins - genetics Glutathione - metabolism Humans Membrane Potential, Mitochondrial - drug effects Mitochondria Mitochondria - drug effects Mitochondria - metabolism Oxidation-Reduction
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