CaMKIIγ regulates the viability and self‐renewal of acute myeloid leukaemia stem‐like cells by the Alox5/NF‐κB pathway

Acute myeloid leukaemia (AML) is a frequently fatal malignant disease of haematopoietic stem and progenitor cells. The molecular and phenotypic characteristics of AML are highly heterogeneous. Our previous study concluded that CaMKIIγ was the trigger of chronic myeloid leukaemia progression from the...

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Veröffentlicht in:	International journal of laboratory hematology 2021-08, Vol.43 (4), p.699-706
Hauptverfasser:	Cheng, Jiang‐Hua, Zhang, Wen‐Jing, Zhu, Jun‐Feng, Cui, Di, Song, Kai‐Di, Qiang, Ping, Mei, Chuan‐Zhong, Nie, Zheng‐Chao, Ding, Bang‐Sheng, Han, Zhong, Ding, Zhi‐En, Zheng, Wei‐Wei
Format:	Artikel
Sprache:	eng
Schlagworte:	acute myeloid leukaemia Acute myeloid leukemia Alox5 Blast crisis CaMKIIγ CD34 antigen Cell lines Chronic myeloid leukemia Hematopoietic stem cells Leukemia Myc protein Progenitor cells Remission self‐renewal Stem cells Viability
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