The transcription factor IRF4 represses proapoptotic BMF and BIM to licence multiple myeloma survival

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Veröffentlicht in:Leukemia 2021-07, Vol.35 (7), p.2114-2118
Hauptverfasser: Fedele, Pasquale L., Liao, Yang, Gong, Jia-nan, Yao, Yuan, van Delft, Mark F., Low, Michael S. Y., Tai, Lin, Herold, Marco J., Jackson, Jacob T., Teh, Charis E., Tan, Tania, O’Reilly, Lorraine A., Tellier, Julie, Grigoriadis, George, Huang, David C. S., Shi, Wei, Nutt, Stephen L., Willis, Simon N.
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container_end_page 2118
container_issue 7
container_start_page 2114
container_title Leukemia
container_volume 35
creator Fedele, Pasquale L.
Liao, Yang
Gong, Jia-nan
Yao, Yuan
van Delft, Mark F.
Low, Michael S. Y.
Tai, Lin
Herold, Marco J.
Jackson, Jacob T.
Teh, Charis E.
Tan, Tania
O’Reilly, Lorraine A.
Tellier, Julie
Grigoriadis, George
Huang, David C. S.
Shi, Wei
Nutt, Stephen L.
Willis, Simon N.
description
doi_str_mv 10.1038/s41375-020-01078-0
format Article
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identifier ISSN: 0887-6924
ispartof Leukemia, 2021-07, Vol.35 (7), p.2114-2118
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source Alma/SFX Local Collection
subjects 38/39
38/91
631/67/1990/804
631/80/82/23
96/2
Ablation
Apoptosis
Cancer
Cancer Research
Cell cycle
CRISPR
Critical Care Medicine
Development and progression
Gene expression
Genetic aspects
Health aspects
Hematology
Intensive
Interferon regulatory factor 4
Internal Medicine
Letter
Medical research
Medicine
Medicine & Public Health
Multiple myeloma
Oncology
Oncology, Experimental
Transcription factors
title The transcription factor IRF4 represses proapoptotic BMF and BIM to licence multiple myeloma survival
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