The transcription factor IRF4 represses proapoptotic BMF and BIM to licence multiple myeloma survival
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Veröffentlicht in: | Leukemia 2021-07, Vol.35 (7), p.2114-2118 |
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container_title | Leukemia |
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creator | Fedele, Pasquale L. Liao, Yang Gong, Jia-nan Yao, Yuan van Delft, Mark F. Low, Michael S. Y. Tai, Lin Herold, Marco J. Jackson, Jacob T. Teh, Charis E. Tan, Tania O’Reilly, Lorraine A. Tellier, Julie Grigoriadis, George Huang, David C. S. Shi, Wei Nutt, Stephen L. Willis, Simon N. |
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doi_str_mv | 10.1038/s41375-020-01078-0 |
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source | Alma/SFX Local Collection |
subjects | 38/39 38/91 631/67/1990/804 631/80/82/23 96/2 Ablation Apoptosis Cancer Cancer Research Cell cycle CRISPR Critical Care Medicine Development and progression Gene expression Genetic aspects Health aspects Hematology Intensive Interferon regulatory factor 4 Internal Medicine Letter Medical research Medicine Medicine & Public Health Multiple myeloma Oncology Oncology, Experimental Transcription factors |
title | The transcription factor IRF4 represses proapoptotic BMF and BIM to licence multiple myeloma survival |
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