B cells in rheumatoid arthritis synovial tissues encode focused antibody repertoires that include antibodies that stimulate macrophage TNF-α production

Rheumatoid arthritis (RA) is characterized by the production of anti-citrullinated protein antibodies (ACPAs). To gain insights into the relationship between ACPA-expressing B cells in peripheral blood (PB) and synovial tissue (ST), we sequenced the B cell repertoire in paired PB and ST samples from...

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Veröffentlicht in:Clinical immunology (Orlando, Fla.) Fla.), 2020-03, Vol.212, p.88-97, Article 108360
Hauptverfasser: Elliott, Serra E., Kongpachith, Sarah, Lingampalli, Nithya, Adamska, Julia Z., Cannon, Bryan J., Blum, Lisa K., Bloom, Michelle S., Henkel, Matthew, McGeachy, Mandy J., Moreland, Larry W., Robinson, William H.
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Sprache:eng
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Zusammenfassung:Rheumatoid arthritis (RA) is characterized by the production of anti-citrullinated protein antibodies (ACPAs). To gain insights into the relationship between ACPA-expressing B cells in peripheral blood (PB) and synovial tissue (ST), we sequenced the B cell repertoire in paired PB and ST samples from five individuals with established, ACPA+ RA. Bioinformatics analysis of paired heavy- and light-chain sequences revealed clonally-related family members shared between PB and ST. ST-derived antibody repertoires exhibited reduced diversity and increased normalized clonal family size compared to PB-derived repertoires. Functional characterization showed that seven recombinant antibodies (rAbs) expressed from subject-derived sequences from both compartments bound citrullinated antigens and immune complexes (ICs) formed using one ST-derived rAb stimulated macrophage TNF-α production. Our findings demonstrate B cell trafficking between PB and ST in subjects with RA and ST repertoires include B cells that encode ACPA capable of forming ICs that stimulate cellular responses implicated in RA pathogenesis.
ISSN:1521-6616
1521-7035
DOI:10.1016/j.clim.2020.108360