miR‐29a attenuates cardiac hypertrophy through inhibition of PPARδ expression

Although cardiac hypertrophy is widely recognized as a risk factor that leads to cardiac dysfunction and, ultimately, heart failure, the complex mechanisms underlying cardiac hypertrophy remain incompletely characterized. The nuclear receptor peroxisome proliferator‐activated receptor δ (PPARδ) is i...

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Veröffentlicht in:	Journal of cellular physiology 2019-08, Vol.234 (8), p.13252-13262
Hauptverfasser:	Zhang, Si, Yin, Zhongnan, Dai, Fei‐Fei, Wang, Hao, Zhou, Meng‐Jiao, Yang, Ming‐Hui, Zhang, Shu‐Feng, Fu, Zhi‐Feng, Mei, Ying‐Wu, Zang, Ming‐Xi, Xue, Lixiang
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Atrial Natriuretic Factor - metabolism Atrial natriuretic peptide cardiac hypertrophy microRNA nuclear receptor PPARδ isoproterenol hydrochloride transcriptional regulation Cardiomegaly - metabolism Congestive heart failure Down-Regulation Gene Expression Regulation - physiology Heart Hypertrophy Isoproterenol Lipid metabolism Lipids Metabolism Mice Mice, Inbred ICR MicroRNAs - metabolism miRNA Myocytes, Cardiac - metabolism Receptors, Cytoplasmic and Nuclear - metabolism Ribonucleic acid Risk analysis Risk factors RNA Therapeutic applications
Online-Zugang:	Volltext
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