Identification of TAF1, HNF4A, and CALM2 as potential therapeutic target genes for liver fibrosis

The molecular mechanism of liver fibrosis caused by hepatitis C virus (HCV) is not clear. The aim of this study is to understand the molecular mechanism of liver fibrosis induced by HCV and to identify potential therapeutic targets for hepatic fibrosis. We analyzed gene expression patterns between h...

Ausführliche Beschreibung

Gespeichert in:

Bibliographische Detailangaben
Veröffentlicht in:	Journal of cellular physiology 2019-06, Vol.234 (6), p.9045-9051
Hauptverfasser:	Ji, Dong, Chen, Guo‐Feng, Wang, Jin‐Cheng, Cao, Li‐Hua, Lu, Fengmin, Mu, Xiao‐Xin, Zhang, Xiao‐Yu, Lu, Xiao‐Jie
Format:	Artikel
Sprache:	eng
Schlagworte:	Bile Biomarkers CALM2 Calmodulin - genetics Calmodulin - metabolism Case-Control Studies Databases, Genetic Fibrosis Gene expression Gene Expression Regulation Gene Regulatory Networks Genes hepatitis B virus Hepatitis C Hepatitis C - complications Hepatitis C - virology hepatitis C virus Hepatocyte nuclear factor 4 Hepatocyte Nuclear Factor 4 - genetics Hepatocyte Nuclear Factor 4 - metabolism Histone Acetyltransferases - genetics Histone Acetyltransferases - metabolism HNF4A Humans Liver Liver - metabolism Liver - pathology Liver - virology Liver Cirrhosis - genetics Liver Cirrhosis - metabolism Liver Cirrhosis - pathology Liver Cirrhosis - virology liver fibrosis Protein Interaction Maps Signal Transduction TAF1 Target recognition TATA-Binding Protein Associated Factors - genetics TATA-Binding Protein Associated Factors - metabolism Therapeutic applications Transcription Factor TFIID - genetics Transcription Factor TFIID - metabolism Viruses
Online-Zugang:	Volltext
Tags:	Tag hinzufügen Keine Tags, Fügen Sie den ersten Tag hinzu!

Schreiben Sie den ersten Kommentar!