Role of corticosterone in immunosuppressive effects of acute ethanol exposure on Toll-like receptor mediated cytokine production

Acute ethanol (EtOH) exposure causes a stress response in humans, nonhuman primates, and rodents. Previous study results indicate that the suppression of some immunological parameters by EtOH is mediated in part or completely by elevated corticosterone concentrations induced by EtOH. However, initia...

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Veröffentlicht in:	Journal of neuroimmune pharmacology 2006-12, Vol.1 (4), p.435-442
Hauptverfasser:	Glover, Mitzi, Pruett, Stephen B
Format:	Artikel
Sprache:	eng
Schlagworte:	Alcohol Drinking - immunology Alcohol Drinking - metabolism Animals Corticosterone - physiology Cytokines Cytokines - biosynthesis Ethanol - administration & dosage Ethanol - toxicity Female Humans Immunosuppressive Agents - administration & dosage Immunosuppressive Agents - toxicity Mice Mice, Inbred C3H Mice, Inbred C57BL Primates Time Factors Toll-Like Receptors - physiology
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