Oxidative stress-modulating drugs have preferential anticancer effects - involving the regulation of apoptosis, DNA damage, endoplasmic reticulum stress, autophagy, metabolism, and migration

To achieve preferential effects against cancer cells but less damage to normal cells is one of the main challenges of cancer research. In this review, we explore the roles and relationships of oxidative stress-mediated apoptosis, DNA damage, ER stress, autophagy, metabolism, and migration of ROS-mod...

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Veröffentlicht in:	Seminars in cancer biology 2019-10, Vol.58, p.109-117
Hauptverfasser:	Tang, Jen-Yang, Ou-Yang, Fu, Hou, Ming-Feng, Huang, Hurng-Wern, Wang, Hui-Ru, Li, Kun-Tzu, Fayyaz, Sundas, Shu, Chih-Wen, Chang, Hsueh-Wei
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Anticancer drugs Antineoplastic Agents - pharmacology Antineoplastic Agents - therapeutic use Autophagy - drug effects Cell Movement - drug effects DNA Damage - drug effects Endoplasmic Reticulum Stress - drug effects Function Humans Oxidative stress Oxidative Stress - drug effects Preferential killing Redox homeostasis
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