Helicobacter pylori cholesterol glucosylation modulates autophagy for increasing intracellular survival in macrophages

Cholesterol‐α‐glucosyltransferase (CGT) encoded by the type 1 capsular polysaccharide biosynthesis protein J (capJ) gene of Helicobacter pylori converts cellular cholesterol into cholesteryl glucosides. H. pylori infection induces autophagy that may increase bacterial survival in epithelial cells. H...

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Veröffentlicht in:	Cellular microbiology 2018-12, Vol.20 (12), p.e12947-n/a
Hauptverfasser:	Lai, Chih‐Ho, Huang, Ju‐Chun, Cheng, Hsin‐Hung, Wu, Meng‐Chen, Huang, Mei‐Zi, Hsu, Hui‐Ying, Chen, Yu‐An, Hsu, Chung‐Yao, Pan, Yi‐Jiun, Chu, Yen‐Ting, Chen, Tsan‐Jan, Wu, Yu‐Fang, Sit, Wei Yang, Liu, Jai‐Shin, Chiu, Ya‐Fang, Wang, Hung‐Jung, Wang, Wen‐Ching
Format:	Artikel
Sprache:	eng
Schlagworte:	Autophagy Bacteria Biosynthesis Capsular polysaccharides Cholesterol cholesterol glucosylation Epithelial cells Glucosides Glucosyltransferase Helicobacter pylori Lipid rafts Lipids Lysosomes Macrophages Phagocytosis Polysaccharides Protein biosynthesis Proteins Rafts Survival
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