Mutagenicity of diesel exhaust particles mediated by cell–particle interaction in mammalian cells

Abstract Diesel exhaust particle (DEP) has been identified as a class 2A human carcinogen and closely related to the increased incidence of respiratory allergy, cardiopulmonary morbidity and mortality, and risk of lung cancer. However, the molecular mechanisms of DEP mutagenicity/carcinogenicity are...

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Veröffentlicht in:	Toxicology (Amsterdam) 2007-01, Vol.229 (1), p.91-100
Hauptverfasser:	Bao, Lingzhi, Chen, Shaopeng, Wu, Lijun, Hei, Tom K, Wu, Yuejin, Yu, Zengliang, Xu, An
Format:	Artikel
Sprache:	eng
Schlagworte:	Ammonium Chloride - pharmacology Animals Biological and medical sciences CD59 Antigens - genetics Cell Line Cell Nucleus - drug effects Cell Nucleus - ultrastructure Cell Size Cell Survival - drug effects Chemical mutagenesis Cricetinae Cytochalasin B - pharmacology Diesel exhaust particles (DEP) Dose-Response Relationship, Drug Emergency Flow Cytometry Humans Human–hamster hybrid (AL) cells Hybrid Cells - drug effects Hybrid Cells - metabolism Hybrid Cells - ultrastructure Inclusion Bodies Light Medical sciences Microscopy, Electron, Scanning Microscopy, Electron, Transmission Mutagenicity Mutagenicity Tests - methods Mutagens - analysis Mutagens - toxicity Particle Size Phagocytosis inhibitors Submitochondrial Particles - drug effects Submitochondrial Particles - ultrastructure Time Factors Toxicology Vehicle Emissions - analysis Vehicle Emissions - prevention & control Vehicle Emissions - toxicity
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