Inactivation of Apc in the mouse prostate causes prostate carcinoma

Alterations of the Wnt/beta-catenin signaling pathway are positively associated with the development and progression of human cancer, including carcinoma of the prostate. To determine the role of activated Wnt/beta-catenin signaling in mouse prostate carcinogenesis, we created a mouse prostate tumor...

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Veröffentlicht in:	Cancer research (Chicago, Ill.) Ill.), 2007-03, Vol.67 (6), p.2490-2496
Hauptverfasser:	BRUXVOORT, Katia J, CHARBONNEAU, Holli M, SIGLER, Robert E, RESAU, James H, SULLIVAN, Ruth, BUSHMAN, Wade, WILLIAMS, Bart O, GIAMBERNARDI, Troy A, GOOLSBY, James C, QIAN, Chao-Nan, ZYLSTRA, Cassandra R, ROBINSON, Daniel R, ROY-BURMAN, Pradip, SHAW, Aubie K, BUCKNER-BERGHUIS, Bree D
Format:	Artikel
Sprache:	eng
Schlagworte:	Alleles Androgens - deficiency Androgens - metabolism Animals Antineoplastic agents beta Catenin - metabolism Biological and medical sciences Cell Nucleus - metabolism Cell Transformation, Neoplastic - genetics Cell Transformation, Neoplastic - metabolism Female Gene Deletion Gene Expression Regulation, Neoplastic Gene Silencing Genes, APC Gynecology. Andrology. Obstetrics Immunohistochemistry Male Male genital diseases Medical sciences Mice Mice, Inbred C57BL Mice, Transgenic Neoplasms, Hormone-Dependent - genetics Neoplasms, Hormone-Dependent - metabolism Nephrology. Urinary tract diseases Organ Specificity Pharmacology. Drug treatments Prostatic Hyperplasia - genetics Prostatic Hyperplasia - metabolism Prostatic Neoplasms - genetics Prostatic Neoplasms - metabolism Tumors Tumors of the urinary system Urinary tract. Prostate gland
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